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机译:NADPH氧化酶2介导的NLRP1炎症组活化涉及在体外的海马神经元中的神经元衰老中
Anhui Med Univ Dept Pharmacol Key Lab Antiinflammatory &
Immunopharmacol Minist Educ Hefei;
Anhui Med Univ Dept Pharmacol Key Lab Antiinflammatory &
Immunopharmacol Minist Educ Hefei;
Anhui Med Univ Dept Pharmacol Key Lab Antiinflammatory &
Immunopharmacol Minist Educ Hefei;
Anhui Med Univ Dept Pharmacol Key Lab Antiinflammatory &
Immunopharmacol Minist Educ Hefei;
Anhui Med Univ Dept Pharmacol Key Lab Antiinflammatory &
Immunopharmacol Minist Educ Hefei;
Anhui Med Univ Dept Pharmacol Key Lab Antiinflammatory &
Immunopharmacol Minist Educ Hefei;
Anhui Med Univ Synthet Lab Basic Med Coll Hefei 230032 Anhui Peoples R China;
Anhui Med Univ Dept Pharmacol Key Lab Antiinflammatory &
Immunopharmacol Minist Educ Hefei;
Anhui Med Univ Dept Pharmacol Key Lab Antiinflammatory &
Immunopharmacol Minist Educ Hefei;
Neuronal senescence; ROS oxidative stress; NADPH oxidase; NLRP1 inflammasome; Neuroinflammation;
机译:NADPH氧化酶2介导的NLRP1炎症组活化涉及在体外的海马神经元中的神经元衰老中
机译:由于在体外海马神经元中的NLRP1炎性信号传导途径的抑制,人参皂甙RG1可防止H2O2诱导的神经元损伤
机译:慢性地塞米松治疗导致海马神经元损伤,因为活化NLRP1炎性炎症
机译:NADPH氧化酶及微胶质激活6-0HDA诱导的多巴胺能神经元变性
机译:神经酰胺代谢调节神经元NADPH氧化酶,影响炎症过程中神经元的存活。
机译:人参皂苷Rg1可以防止H2O2诱导的神经元损伤这是由于在体外抑制海马神经元中的NLRP1炎性体信号通路
机译:由于在海马神经元中的NLRP1炎性信号传导途径抑制,人参皂甙RG1可防止H2O2诱导的神经元损伤.Hipocampal NeuronsIn�vitro