Lack of the extracellular matrix proteoglycan lumican leads to cardiac dysfunction in response to pressure overload

Mohammadzadeh N.; Engebretsen K.; Lunde I.; Sjaastad I.; Strand M.; Marstein H.; Aronsen J.; Nygard S.; Skrbic B.; Bandlien C.; Andenaes K.; Chakravarti S.; Christensen G.; Tonnessen T.

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Lack of the extracellular matrix proteoglycan lumican leads to cardiac dysfunction in response to pressure overload

机译：缺乏细胞外基质蛋白多糖蛋白，伴随压力过载的心脏功能障碍

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《Acta physiologica》 |2017年第711期|共1页
作者
Mohammadzadeh N.; Engebretsen K.; Lunde I.; Sjaastad I.; Strand M.; Marstein H.; Aronsen J.; Nygard S.; Skrbic B.; Bandlien C.; Andenaes K.; Chakravarti S.; Christensen G.; Tonnessen T.;
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Oslo Univ Hosp Expt Med Res Inst Ulleval Sykehus Norway;

Oslo Univ Hosp Expt Med Res Inst Ulleval Sykehus Norway;

Oslo Univ Hosp Expt Med Res Inst Ulleval Sykehus Norway;

Oslo Univ Hosp Expt Med Res Inst Ulleval Sykehus Norway;

Oslo Univ Hosp Expt Med Res Inst Ulleval Sykehus Norway;

Oslo Univ Hosp Expt Med Res Inst Ulleval Sykehus Norway;

Oslo Univ Hosp Expt Med Res Inst Ulleval Sykehus Norway;

Oslo Univ Hosp Expt Med Res Inst Ulleval Sykehus Norway;

Oslo Univ Hosp Expt Med Res Inst Ulleval Sykehus Norway;

Oslo Univ Hosp Expt Med Res Inst Ulleval Sykehus Norway;

Oslo Univ Hosp Expt Med Res Inst Ulleval Sykehus Norway;

Johns Hopkins Univ Baltimore MD USA;

Oslo Univ Hosp Expt Med Res Inst Ulleval Sykehus Norway;

Oslo Univ Hosp Expt Med Res Inst Ulleval Sykehus Norway;

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正文语种 eng
中图分类人体生理学;
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1. Lack of the extracellular matrix proteoglycan lumican leads to cardiac dysfunction in response to pressure overload [J] . Mohammadzadeh N., Engebretsen K., Lunde I., Acta physiologica . 2017,第Suppla711期

机译：缺乏细胞外基质蛋白多糖蛋白，伴随压力过载的心脏功能障碍
2. Moderate Loss of the Extracellular Matrix Proteoglycan Lumican Attenuates Cardiac Fibrosis in Mice Subjected to Pressure Overload [J] . Mohammadzadeh Naiyereh, Melleby Arne Olav, Palmero Sheryl, Cardiology . 2020,第3期

机译：细胞外基质的中度损失蛋白多糖蛋白伴随于对压力过载进行压力过载的小鼠中的心肌纤维化
3. Lack of tissue inhibitor of metalloproteinases 2 leads to exacerbated left ventricular dysfunction and adverse extracellular matrix remodeling in response to biomechanical stress. [J] . Kandalam V, Basu R, Moore L, Circulation: An Official Journal of the American Heart Association . 2011,第19期

机译：缺乏金属蛋白酶2的组织抑制剂会导致左心室功能障碍加重，并对生物力学应力产生不良的细胞外基质重塑。
4. The examination of extracellular matrix proteoglycans and growth factors in decellularized tendon slices [C] . Liang-ju Ning, Yi Zhang, Xiao-He Chen, World biomaterials congress . 2012

机译：去细胞肌腱切片中细胞外基质蛋白聚糖和生长因子的检查
5. Effects of age and immune dysfunction on the cardiac extracellular matrix and diastolic function. [D] . Alwardt, Cory M. 2005

机译：年龄和免疫功能障碍对心脏细胞外基质和舒张功能的影响。
6. The extracellular matrix proteoglycan lumican improves survival and counteracts cardiac dilatation and failure in mice subjected to pressure overload [O] . Naiyereh Mohammadzadeh, Ida G. Lunde, Kine Andenæs, -1

机译：细胞外基质蛋白聚糖Lumican改善了压力超负荷小鼠的存活率并抵消了心脏的扩张和衰竭
7. The extracellular matrix proteoglycan lumican improves survival and counteracts cardiac dilatation and failure in mice subjected to pressure overload [O] . Naiyereh Mohammadzadeh, Ida G. Lunde, Kine Andenæs, 2019

机译：细胞外基质蛋白多糖伴娘改善生存率并抵消对压力过载的小鼠的心脏扩张和失效

Lack of the extracellular matrix proteoglycan lumican leads to cardiac dysfunction in response to pressure overload

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