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机译:GNA的升高表达促进了通过PI3K / AKT / SNAIL1 / E-Cadherin轴促进乳腺癌细胞增殖和迁移
Chongqing Med Univ Dept Endocrine &
Breast Surg Affiliated Hosp 1 1 Youyi Rd Chongqing 400016;
First Peoples Hosp Yibin Dept Breast Surg Yibin Sichuan Peoples R China;
First Peoples Hosp Yibin Dept Breast Surg Yibin Sichuan Peoples R China;
First Peoples Hosp Yibin Dept Breast Surg Yibin Sichuan Peoples R China;
First Peoples Hosp Yibin Dept Breast Surg Yibin Sichuan Peoples R China;
Chongqing Med Univ Dept Endocrine &
Breast Surg Affiliated Hosp 1 1 Youyi Rd Chongqing 400016;
GNAS; Breast cancer; PI3K; AKT signaling; Cell proliferation; Cell migration; Metastasis;
机译:GNA的升高表达促进了通过PI3K / AKT / SNAIL1 / E-Cadherin轴促进乳腺癌细胞增殖和迁移
机译:长链非编码SOX21-AS1通过PI3K / AKT信号通路促进乳腺癌细胞的增殖和迁移
机译:通过MIR-378A-3P / GALNT3轴激活PI3K / AKT通路,LNCRNA PSMA3-AS1促进卵巢癌中的细胞增殖,迁移和侵袭
机译:Ang-2通过PI3K / Akt途径促进SW1116结肠癌细胞的增殖
机译:DLX4同源蛋白通过上调VEGFA促进ER阴性乳腺癌细胞中的PI3K / Akt抗凋亡途径。
机译:长链非编码SOX21-AS1通过PI3K / AKT信号通路促进乳腺癌细胞的增殖和迁移
机译:GNA的升高表达促进了通过PI3K / AKT / SNAIL1 / E-Cadherin轴促进乳腺癌细胞增殖和迁移