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机译:胃肠肥大细胞的脱落有助于小鼠的肝缺血再灌注损伤
Tongji Univ Shanghai Peoples Hosp 10 Sch Med Dept Gen Surg 301 Middle Yanchang Rd Shanghai;
Tongji Univ Shanghai Peoples Hosp 10 Sch Med Dept Obstet &
Gynecol 301 Middle Yanchang Rd;
Tongji Univ Shanghai Peoples Hosp 10 Sch Med Dept Gen Surg 301 Middle Yanchang Rd Shanghai;
Tongji Univ Shanghai Peoples Hosp 10 Sch Med Dept Gen Surg 301 Middle Yanchang Rd Shanghai;
Tongji Univ Shanghai Peoples Hosp 10 Sch Med Dept Gen Surg 301 Middle Yanchang Rd Shanghai;
Tongji Univ Shanghai Peoples Hosp 10 Sch Med Dept Gen Surg 301 Middle Yanchang Rd Shanghai;
Tongji Univ Shanghai Peoples Hosp 10 Sch Med Dept Gen Surg 301 Middle Yanchang Rd Shanghai;
Tongji Univ Shanghai Peoples Hosp 10 Sch Med Dept Gen Surg 301 Middle Yanchang Rd Shanghai;
Nanjing Univ Dept Cardiol Jiangsu Prov Hosp Sch Med Nanjing 210029 Jiangsu Peoples R China;
Tongji Univ Shanghai Peoples Hosp 10 Sch Med Dept Gen Surg 301 Middle Yanchang Rd Shanghai;
机译:胃肠肥大细胞的脱落有助于小鼠的肝缺血再灌注损伤
机译:肥大细胞脱颗粒促进大鼠肝脏缺血再灌注损伤
机译:缺血前肥大细胞脱粒减少了犬小肠的缺血-再灌注损伤。
机译:抑制常规心脏肥大细胞脱粒可能预防缺血性和再灌注诱导的心肌损伤
机译:骨内源性,腹膜内和静脉内转移到遗传上缺乏乳腺的W / Wv小鼠后,骨髓来源的肥大细胞的命运证据表明,培养的肥大细胞可以使结缔组织类型和粘膜肥大同时上升
机译:胃肠肥大细胞的去颗粒化导致小鼠肝缺血-再灌注损伤
机译:肥大细胞通过胶酶/肥大细胞蛋白酶4依赖性机制有助于含有博来霉素诱导的小鼠肺炎和损伤