机译:巴黎皂苷诱导的自噬通过AKT / MTOR信号通路促进乳腺癌细胞凋亡
Jinan Univ Guangzhou Jinan Biomed Res &
Dev Ctr Coll Life Sci &
Technol Guangdong Prov Key Lab;
Jinan Univ Coll Pharm Guangzhou 510632 Guangdong Peoples R China;
Jinan Univ Guangzhou Jinan Biomed Res &
Dev Ctr Coll Life Sci &
Technol Guangdong Prov Key Lab;
Jinan Univ Guangzhou Jinan Biomed Res &
Dev Ctr Coll Life Sci &
Technol Guangdong Prov Key Lab;
Jinan Univ Coll Pharm Guangzhou 510632 Guangdong Peoples R China;
Jinan Univ Guangzhou Jinan Biomed Res &
Dev Ctr Coll Life Sci &
Technol Guangdong Prov Key Lab;
Jinan Univ Guangzhou Jinan Biomed Res &
Dev Ctr Coll Life Sci &
Technol Guangdong Prov Key Lab;
Jinan Univ Guangzhou Jinan Biomed Res &
Dev Ctr Coll Life Sci &
Technol Guangdong Prov Key Lab;
Jinan Univ Guangzhou Jinan Biomed Res &
Dev Ctr Coll Life Sci &
Technol Guangdong Prov Key Lab;
Jinan Univ Guangzhou Jinan Biomed Res &
Dev Ctr Coll Life Sci &
Technol Guangdong Prov Key Lab;
Guangdong Inst Drug Control Guangzhou 510632 Guangdong Peoples R China;
Jinan Univ Guangzhou Jinan Biomed Res &
Dev Ctr Coll Life Sci &
Technol Guangdong Prov Key Lab;
Paris saponin; Apoptosis; Autophagy;
机译:巴黎皂苷诱导的自噬通过AKT / MTOR信号通路促进乳腺癌细胞凋亡
机译:研究性Aurora激酶A抑制剂alisertib(MLN8237)通过p38 MAPK和Akt / mTOR信号通路诱导人乳腺癌细胞G2 / M阻滞,凋亡和自噬
机译:通过Akt / mtor和P38信号传导途径诱导细胞凋亡和自噬使乳腺癌细胞(DOX)的含量敏感性提高了乳腺癌细胞的敏感性
机译:阳离子聚苯乙烯纳米球通过抑制Akt / mTOR和激活巨噬细胞和上皮细胞中的AMPK信号通路来诱导自噬
机译:DLX4同源蛋白通过上调VEGFA促进ER阴性乳腺癌细胞中的PI3K / Akt抗凋亡途径。
机译:缩回:内质网应激的激活通过抑制PI3K / AKT / mTOR信号通路促进人小细胞肺癌细胞的自噬和凋亡并逆转化学耐药性
机译:收缩:内质网胁迫的激活促进了自噬和凋亡,通过抑制PI3K / AKT / MTOR信号传导途径来逆转人小细胞肺癌细胞的化学感受