PAX5, NOTCH3, CBFB, and ACD drive an activated RAS pathway and monosomy 7 to B-ALL and AML in donor cell leukemia

Assi Rita; Mahfouz Rami; Owen Renius; Gunthorpe Martha; Chehab Farid F.; Bazarbachi Ali

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PAX5, NOTCH3, CBFB, and ACD drive an activated RAS pathway and monosomy 7 to B-ALL and AML in donor cell leukemia

机译：PAX5，NOTCH3，CBFB和ACD驱动活化的RAS途径和单体7至B-全部和AML在供体细胞白血病中

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《Bone marrow transplantation》 |2019年第7期|共5页
作者
Assi Rita; Mahfouz Rami; Owen Renius; Gunthorpe Martha; Chehab Farid F.; Bazarbachi Ali;
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作者单位

Univ Texas MD Anderson Canc Ctr Dept Leukemia Houston TX 77030 USA;

Amer Univ Beirut Dept Pathol &

Lab Med Med Ctr Beirut Lebanon;

Nichols Inst Quest Diagnost San Juan Capistrano CA USA;

Univ Calif San Francisco Dept Lab Med San Francisco CA 94143 USA;

Univ Calif San Francisco Dept Lab Med San Francisco CA 94143 USA;

Amer Univ Beirut Dept Internal Med Med Ctr Beirut Lebanon;

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原文格式 PDF
正文语种 eng
中图分类治疗学;
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1. PAX5, NOTCH3, CBFB, and ACD drive an activated RAS pathway and monosomy 7 to B-ALL and AML in donor cell leukemia [J] . Assi Rita, Mahfouz Rami, Owen Renius, Bone marrow transplantation . 2019,第7期

机译：PAX5，NOTCH3，CBFB和ACD驱动活化的RAS途径和单体7至B-全部和AML在供体细胞白血病中
2. Donor KIR-Activating Genes and Patient KIR Ligand Status Modulate Leukemia Relapse and Leukemia Free Survival after Haploidentical Hematopoietic Stem Cell Transplantation in a Cohort Dominated by High Risk AML and ALL Patients in China [J] . Jing-bo Wang, Xiang-jun Liu, Wei-jie Zhang Biology of blood and marrow transplantation: journal of the American Society for Blood and Marrow Transplantation . 2018,第3期

机译：None
3. Donor KIR-Activating Genes and Patient KIR Ligand Status Modulate Leukemia Relapse and Leukemia Free Survival after Haploidentical Hematopoietic Stem Cell Transplantation in a Cohort Dominated by High Risk AML and ALL Patients in China [J] . Jing-bo Wang, Xiang-jun Liu, Wei-jie Zhang Biology of blood and marrow transplantation: journal of the American Society for Blood and Marrow Transplantation . 2018,第3期

机译：供体KIR激活基因和患者KIR配体状态调节白血病复发和白血病在杂志造血干细胞移植后的白血病复发和白血病免费生存，以高风险AML和中国的所有患者为主。
4. Detection of de novo IGHV mutations by ultra-deep sequencing from in vitro activated B-cell chronic lymphocytic leukemia cells: Evidence for activation-induced deaminase function [C] . Chu C.C., Patten P.E.M., MacCarthy T, International Congress of Immunology. . 2014

机译：通过来自体外活化B细胞慢性淋巴细胞白血病细胞超深序的De Novo IghV突变检测：活化诱导的脱氨酶功能的证据
5. Activation of chloride channels with the anti-parasitic agent ivermectin induces membrane hyperpolarization and cell death in leukemia cells. [D] . Sharmeen, Sumaiya. 2010

机译：用抗寄生虫药伊维菌素激活氯离子通道可诱导白血病细胞膜超极化和细胞死亡。
6. AML associated oncofusion proteins PML-RARA AML1-ETO and CBFB-MYH11 target RUNX/ETS-factor binding sites to modulate H3ac levels and drive leukemogenesis [O] . Abhishek A. Singh, Amit Mandoli, Koen H.M. Prange, 2017

机译：AML相关的肿瘤融合蛋白PML-RARAAML1-ETO和CBFB-MYH11靶向RUNX / ETS因子结合位点以调节H3ac水平并驱动白血病发生
7. Donor KIR-Activating Genes and Patient KIR Ligand Status Modulate Leukemia Relapse and Leukemia Free Survival after Haploidentical Hematopoietic Stem Cell Transplantation in a Cohort Dominated by High Risk AML and ALL Patients in China [O] . Jing-bo Wang, Xiang-jun Liu, Wei-jie Zhang 2018

机译：供体KIR激活基因和患者KIR配体状态调节白血病复发和白血病在杂志造血干细胞移植后的白血病复发和白血病免费生存，以高风险AML和中国的所有患者为主。

PAX5, NOTCH3, CBFB, and ACD drive an activated RAS pathway and monosomy 7 to B-ALL and AML in donor cell leukemia

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