机译:缺氧选择性的激活激素类激酶的稳定化:潜在的治疗途径,用于异位骨化的预防
Texas A&
M Univ Coll Dent Dept Biomed Sci Dallas TX 75246 USA;
Texas A&
M Univ Coll Dent Dept Biomed Sci Dallas TX 75246 USA;
Texas A&
M Univ Coll Dent Dept Biomed Sci Dallas TX 75246 USA;
Texas A&
M Univ Coll Dent Dept Biomed Sci Dallas TX 75246 USA;
Texas A&
M Univ Coll Dent Dept Biomed Sci Dallas TX 75246 USA;
Bone morphogenetic protein; BMP; Protein kinase; Allosteric; R-spine; alpha C-beta 4 loop; ACVR1; ALK2; FKBP12; BMPRII; Heterotopic ossification; Fibrodysplasia ossificans progressiva; FOP; Hypoxia; Hypoxic pH; Pharmacological chaperone; Selective degrader; Hydrophobic tagging; PROTACs; Small molecule kinase inhibitor;
机译:活素受体样激酶的缺氧选择性变构稳定化:潜在的治疗途径,用于异位骨化的预防
机译:破骨细胞在骨增生性纤维增生症小鼠中与增生相关的激活素样激酶2突变增强的异位骨化中的作用
机译:Fibrodysplasia Ossificans进展相关的活化激活素样激酶信号传导在肌肉组织中的异位骨化期间增强了破骨细胞形成
机译:良性疾病放射治疗的随机临床试验:异位骨化预防域的范式
机译:RNA干扰预防战斗创伤引起的异位骨化。
机译:通过抑制激活素受体样激酶2功能靶向异位骨化
机译:激活素受体样激酶1抑制人微血管内皮细胞迁移:JNK和ERK的潜在作用。