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机译:PD-1封闭和endoStar对Lewis肺癌小鼠模型PI3K / AKT / MTOR介导的自噬和血管生成的协同作用
Shandong Univ Hosp 2 Dept Pharm Jinan Peoples R China;
Shandong Univ Hosp 2 Dept Thorac Surg Jinan Peoples R China;
Shandong Prov Hosp Dept Thorac Surg Jinan Peoples R China;
YantaiYuhuangding Hosp Dept Thorac Surg Yantai Peoples R China;
Shandong Univ Hosp 2 Dept Thorac Surg Jinan Peoples R China;
Shandong Univ Hosp 2 Dept Thorac Surg Jinan Peoples R China;
Shandong Univ Key Lab Chest Canc Jinan Peoples R China;
anti-PD-1; Endostar; Synergy; Autophagy; NSCLC;
机译:PD-1封闭和endoStar对Lewis肺癌小鼠模型PI3K / AKT / MTOR介导的自噬和血管生成的协同作用
机译:Plumbagin通过p38 MAPK和PI3K / Akt / mTOR介导的人舌鳞状细胞癌细胞G2 / M阻滞,凋亡和自噬
机译:抗血管生成治疗克服了VEGFA过表达小鼠肿瘤模型中PD-1 / PD-L1阻断的先天性抗性
机译:Endostar通过调节HIF-1α增强对刘易斯肺癌的放射反应
机译:NTCU小鼠肺鳞癌模型的表征:维生素D的作用
机译:Endostar通过影响能量代谢并减轻Lewis肺癌小鼠模型的肿瘤微环境来增强放射线的抗肿瘤作用
机译:EZOX A通过P38 MAPK-and PI3K / AKT / MTOR介导的小鼠脾细胞介导的途径诱导G1骤停和自噬