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机译:二甲双胍通过IGF-IR / PI3K / AKT / M-TOR通道抑制类风湿性关节炎成纤维细胞样Synociytes的增殖
Southern Med Univ Zhujiang Hosp Dept Orthoped Guangzhou 510282 Guangdong Peoples R China;
Southern Med Univ Zhujiang Hosp Dept Orthoped Guangzhou 510282 Guangdong Peoples R China;
Univ South China Affiliated Hosp 2 Dept Orthoped Hengyang 421001 Hunan Peoples R China;
Southern Med Univ Zhujiang Hosp Dept Orthoped Guangzhou 510282 Guangdong Peoples R China;
Southern Med Univ Zhujiang Hosp Dept Orthoped Guangzhou 510282 Guangdong Peoples R China;
Southern Med Univ Zhujiang Hosp Dept Orthoped Guangzhou 510282 Guangdong Peoples R China;
Southern Med Univ Zhujiang Hosp Dept Orthoped Guangzhou 510282 Guangdong Peoples R China;
Southern Med Univ Zhujiang Hosp Dept Orthoped Guangzhou 510282 Guangdong Peoples R China;
Metformin; Proliferation; Cell cycle; Rheumatoid arthritis; Fibroblast-like synoviocytes; IGF-IR /PI3K/AKT/ mTOR pathway;
机译:二甲双胍通过IGF-IR / PI3K / AKT / M-TOR通道抑制类风湿性关节炎成纤维细胞样Synociytes的增殖
机译:FOXC1通过PI3K / AKT信号通路促进类风湿性关节炎中成纤维细胞样Synociocytes的增殖
机译:Trichostatin A是组蛋白脱乙酰基酶的抑制剂,可通过PI3K / Akt信号传导抑制低氧类风湿关节炎成纤维细胞样滑膜细胞的活力和侵袭性
机译:Ang-2通过PI3K / Akt途径促进SW1116结肠癌细胞的增殖
机译:IL-27和尿酸结晶对类风湿关节炎成纤维细胞样滑膜细胞活化以及青藤碱和凉庙散对TNF-α激活的成纤维细胞样滑膜细胞的抗炎活性。
机译:MiR-26a-5p通过调节PTEN / PI3K / AKT途径增强类风湿关节炎中成纤维样滑膜细胞的增殖侵袭和凋亡抗性
机译:TrichostaTina,组蛋白脱乙酰酶的抑制剂,抑制缺氧类风湿性关节炎成纤维细胞的活力和侵袭性通过PI3K / AKT信号传导