机译:Diquat诱导的氧化应激增加肠道渗透性,损害线粒体功能,并在仔猪中触发肠球菌
Zhejiang Univ Anim Sci Coll Key Lab Mol Anim Nutr Minist Educ Hangzhou 310058 Zhejiang Peoples R China;
Zhejiang Univ Anim Sci Coll Key Lab Mol Anim Nutr Minist Educ Hangzhou 310058 Zhejiang Peoples R China;
Zhejiang Univ Anim Sci Coll Key Lab Mol Anim Nutr Minist Educ Hangzhou 310058 Zhejiang Peoples R China;
Zhejiang Univ Anim Sci Coll Key Lab Mol Anim Nutr Minist Educ Hangzhou 310058 Zhejiang Peoples R China;
Zhejiang Univ Anim Sci Coll Key Lab Mol Anim Nutr Minist Educ Hangzhou 310058 Zhejiang Peoples R China;
Zhejiang Univ Anim Sci Coll Key Lab Mol Anim Nutr Minist Educ Hangzhou 310058 Zhejiang Peoples R China;
Zhejiang Univ Anim Sci Coll Key Lab Mol Anim Nutr Minist Educ Hangzhou 310058 Zhejiang Peoples R China;
intestinal barrier function; mitochondrial function; mitophagy; oxidative stress;
机译:Diquat诱导的氧化应激增加肠道渗透性,损害线粒体功能,并在仔猪中触发肠球菌
机译:断奶破坏了肠道抗氧化剂状态,损害肠道屏障和线粒体功能,并触发仔猪中的含量
机译:低出生体重仔猪的胎盘容易受到氧化应激,线粒体功能障碍和血管生成受损的伤害
机译:餐后高糖血症引起的氧化应激可通过增加组织多普勒和波强度来评估2型糖尿病非高血压患者的心肌能量需求,从而损害心脏/血管功能。
机译:表征线粒体蛋白酶PARL在线粒体和线粒体质量控制中的功能
机译:敌草快引起的氧化应激会增加肠道通透性损害线粒体功能并触发仔猪的线粒体吞噬
机译:Diquat诱导的氧化应激增加肠道渗透性,损害线粒体功能,并在仔猪中触发MINOCHAGY