机译:通过靶向PI3K / AKT途径抑制HIF-1α/ VEGF表达,Gambogic酸改善糖尿病诱导的增殖性视网膜病变
Department of Ophthalmology The First Affiliated Hospital of China Medical University;
Department of Ophthalmology The Third Affiliated Hospital of Jinzhou Medical University;
Department of Ophthalmology The Third Affiliated Hospital of Jinzhou Medical University;
Department of Ophthalmology The Third Affiliated Hospital of Jinzhou Medical University;
Department of Ophthalmology The First Affiliated Hospital of China Medical University;
Gambogic acid; Diabetic retinopathy; Angiogenesis; HIF-1α; VEGF; PI3K/AKT;
机译:通过靶向PI3K / AKT途径抑制HIF-1α/ VEGF表达,Gambogic酸改善糖尿病诱导的增殖性视网膜病变
机译:Genistein通过抑制PI3K / AKT / HIF-1α/ VEGF和NF-Kappa B / COX-2信号传导途径降低A549细胞活力
机译:吉姆酸主要通过PI3K / AKT和ERK信号通路的抑制表现出恶性黑色素瘤的抗转移活性
机译:基于透明质酸的多价VEGF拮抗剂抑制糖尿病性视网膜病变中的致病性血管生成。
机译:BP1通过激活VEGF调节PI3K / Akt途径。
机译:蛋白激酶C Delta的抑制通过抑制PI3K / Akt / mTOR / HIF-1 Alpha / VEGF途径减轻过敏性气道炎症
机译:Nogo-B以自分泌方式通过VEGF / pI3K / akt途径促进增殖性糖尿病视网膜病变中的血管生成