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机译:雷帕霉素诱导缓解脑缺血和通过MTORC1 / ATG13 / ULK1信号通路再灌注急性肾损伤的自噬
Wuhan Univ Renmin Hosp Dept Urol 99 Zhang Zhi Dong Rd Wuhan 430060 Hubei Peoples R China;
Wuhan Univ Renmin Hosp Dept Urol 99 Zhang Zhi Dong Rd Wuhan 430060 Hubei Peoples R China;
Wuhan Univ Renmin Hosp Dept Neurosurg Wuhan 430060 Hubei Peoples R China;
Anhui Med Univ Affiliated Hosp 1 Dept Urol Hefei 230032 Anhui Peoples R China;
Anhui Med Univ Affiliated Hosp 1 Dept Urol Hefei 230032 Anhui Peoples R China;
Wuhan Univ Renmin Hosp Dept Urol 99 Zhang Zhi Dong Rd Wuhan 430060 Hubei Peoples R China;
cerebral ischemia-reperfusion; acute kidney injury; autophagy; apoptosis; inflammation;
机译:雷帕霉素诱导缓解脑缺血和通过MTORC1 / ATG13 / ULK1信号通路再灌注急性肾损伤的自噬
机译:MicroRNA-338-5P通过靶向组织生长因子通过腺苷5'-单磷酸活性的蛋白激酶/哺乳动物靶向雷帕霉素信号传导途径来减轻脑缺血/再灌注损伤
机译:Empagliflozin通过HIF-1α/ VEGF信号通路缓解通过脑缺血/再灌注损伤诱导的神经元细胞凋亡
机译:全局磷酸化分析揭示了心脏缺血/再灌注损伤期间由成纤维细胞生长因子2(FGF2)引起的心脏保护信号传导的途径
机译:Mitofusin 2介导Sirtuin 1诱导自噬以抑制肝脏缺血/再灌注损伤。
机译:生物晶素A通过抑制内质网胁迫诱导的细胞凋亡和体内和体外P38MAPK信号通路来缓解脑缺血/再灌注损伤
机译:雷帕霉素诱导缓解脑缺血和通过MTORC1 / ATG13 / ULK1信号通路再灌注急性肾损伤的自噬