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首页> 外文期刊>Transplantation Proceedings >Complete neurological recovery from fulminant hepatic failure with subarachnoid hemorrhage by living donor liver transplantation: a case report.
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Complete neurological recovery from fulminant hepatic failure with subarachnoid hemorrhage by living donor liver transplantation: a case report.

机译:通过活体供体肝移植从暴发性肝衰竭合并蛛网膜下腔出血完全神经恢复:一例报告。

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A 29-year-old man was referred to our hospital with fulminant hepatic failure (FHF) and stage III hepatic coma (somnolence and confusion). Living donor liver transplantation (LDLT) was planned for 2 days after admission to our hospital. However, on the day after admission, he lapsed into stage IV hepatic coma: no right reflexes and no response to pain stimuli. Emergency cranial computed tomography revealed a subarachnoid hemorrhage (SAH), but no aneurysm was seen on magnetic resonance angiography. We speculated that the cause of the SAH may have been bleeding of intracranial veins secondary to coagulopathy and overextension of a vein due to brain edema. We considered that only LDLT could improve the coagulopathy and brain edema. The patient recovered consciousness on postoperative day (POD) 2 and was finally discharged from the hospital without neurological deficit on POD 85. This case suggested that SAH is not a prohibiting factor for LDLT in an FHF patient if the cause of the SAH is venous bleeding.
机译:一名29岁的男子因暴发性肝衰竭(FHF)和III期肝昏迷(嗜睡和神志不清)被转诊到我们医院。计划在我院住院两天后进行活体供体肝移植(LDLT)。然而,在入院后的第二天,他陷入了IV期肝昏迷:无右反射,对疼痛刺激无反应。紧急颅脑计算机断层扫描显示蛛网膜下腔出血(SAH),但在磁共振血管造影上未见动脉瘤。我们推测SAH的病因可能是继发于凝血病的颅内静脉出血和由于脑水肿引起的静脉过度延伸。我们认为只有LDLT才能改善凝血功能和脑水肿。该患者在术后第2天恢复了意识,并最终出院,但POD 85并未出现神经系统缺陷。该病例表明,如果SAH的原因是静脉出血,则SAH不是FHF患者LDLT的禁忌因素。 。

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