首页> 外文期刊>The Journal of hand surgery, European volume >Relative contribution of tissue oedema and the presence of an A2 pulley to resistance to flexor tendon movement: An in vitro and in vivo study
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Relative contribution of tissue oedema and the presence of an A2 pulley to resistance to flexor tendon movement: An in vitro and in vivo study

机译:组织水肿和A2滑轮对屈肌腱运动抵抗的相对贡献:一项体外和体内研究

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摘要

Increases in the resistance to tendon during movement may lead to rupture or gapping of the repaired tendon. The relative contribution of these factors-tissue oedema, tendon oedema, and the intact A2 pulley-to resistance to tendon gliding is unclear. In in vitro chicken models, we created oedema in subcutaneous tissue or tendon. The work of digital flexion (WOF) increased significantly after creation of oedema in those tissues. WOF decreased significantly after division of the A2 pulley. Preservation of the integrity of the A2 pulley significantly increased WOF at post-operative weeks 1 and 2 in the in vivo chicken model; increases in WOF were greater than those due to the presence of oedematous subcutaneous tissue. Pulley division at the time of surgery reduced WOF more drastically than the removal of volar subcutaneous tissue. Presence of an intact A2 pulley adds greater resistance to the movement of the repaired flexor tendon than volar oedematous subcutaneous tissue.
机译:运动过程中对肌腱的抵抗力增加可能会导致修复的肌腱破裂或裂开。这些因素(组织水肿,肌腱水肿和完整的A2滑轮对肌腱滑行的抵抗力)的相对贡献尚不清楚。在体外鸡模型中,我们在皮下组织或肌腱中产生了水肿。在这些组织中产生水肿后,数字屈曲(WOF)的工作显着增加。分割A2滑轮后,WOF显着下降。在体内鸡模型中,保留A2滑轮的完整性可显着增加术后1和2周的WOF。由于存在水肿皮下组织,WOF的增加大于那些。与去除掌侧皮下组织相比,外科手术时的滑轮分割可显着降低WOF。完整的A2滑轮的存在比手掌水肿的皮下组织增加了对修复的屈肌腱运动的抵抗力。

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