机译:GDE2通过糖基磷脂酰肌醇固定的RECK促进神经发生。
Solomon Snyder Department of Neuroscience, School of Medicine, Johns Hopkins University, PCTB1004, 725 N Wolfe Street, Baltimore, HAD 21205, USA.;
Solomon Snyder Department of Neuroscience, School of Medicine, Johns Hopkins University, PCTB1004, 725 N Wolfe Street, Baltimore, HAD 21205, USA.;
Solomon Snyder Department of Neuroscience, School of Medicine, Johns Hopkins University, PCTB1004, 725 N Wolfe Street, Baltimore, HAD 21205, USA.;
Solomon Snyder Department of Neuroscience, School of Medicine, Johns Hopkins University, PCTB1004, 725 N Wolfe Street, Baltimore, HAD 21205, USA.;
Department of Pharma-cology and Molecular Sciences, School of Medicine, Johns Hopkins University, WBSB 301A, 725 N Wolfe Street Baltimore,MD 21205, USA;
Solomon Snyder Department of Neuroscience, School of Medicine, Johns Hopkins University, PCTB1004, 725 N Wolfe Street, Baltimore, HAD 21205, USA;
机译:Prdx4是一种特定于隔室的H2O2传感器,可通过控制GDE2的表面表达来调节神经发生
机译:Prdx4是一种隔室特异性H 2 sub> O 2 sub>传感器,通过控制GDE2的表面表达来调节神经发生
机译:GDE2的序列依赖性运输和活性,促进神经元分化的GPI特异性磷脂酶
机译:通过裂解无线金属碳Σ - 键的裂解促进骨灰磷酸硼胺的骨骼重排
机译:密度泛函理论研究金属离子促进和碱促进的硫代磷酸酯裂解反应机理以及镧系元素离子催化剂用于氨基酸酯的动力学拆分。
机译:GDE2促进通过RECK的糖基锚裂解神经发生
机译:PRDX4是一种特定于隔室的H2O2传感器,通过控制GDE2的表面表达来调节神经发生