机译:去甲肾上腺素转运蛋白基因的多态性改变启动子活性,并与注意力缺陷多动障碍相关
Harvard Univ, McLean Hosp, Sch Med, Mol Neurobiol Lab, Belmont, MA 02478 USA;
Harvard Univ, McLean Hosp, Sch Med, Dev Psychopharmacol Lab, Belmont, MA 02478 USA;
Harvard Univ, McLean Hosp, Sch Med, Mol Pharmacol Lab, Belmont, MA 02478 USA;
Vanderbilt Univ, Med Ctr, Dept Pharmacol, Nashville, TN 37232 USA;
Vanderbilt Univ, Med Ctr, Ctr Mol Neurosci, Nashville, TN 37232 USA;
Sungkyunkwan Univ, Sch Med, Samsung Med Ctr, Dept Psychiat, Seoul 135170, South Korea;
Emory Univ, Dept Psychol, Atlanta, GA 30322 USA;
Vanderbilt Univ, Autonom Dysfunct Ctr, Dept Med, Nashville, TN 37232 USA;
Vanderbilt Univ, Autonom Dysfunct Ctr, Dept Pharmacol, Nashville, TN 37232 USA;
Vanderbilt Univ, Autonom Dysfunct Ctr, Dept Neurol, Nashville, TN 37232 USA;
Snail family; E2-box; slug; scratch; BETA-HYDROXYLASE GENE; NO EVIDENCE; NORADRENALINE TRANSPORTER; TRANSCRIPTION FACTORS; K-SADS; CHILDREN; LINKAGE; ADHD; SCHIZOPHRENIA; TACHYCARDIA;
机译:Norepinephrine转运蛋白基因多态性在韩国人口中的注意力/多动障碍中
机译:去甲肾上腺素转运蛋白多态性对哌醋甲酯引起的注意力缺陷多动障碍神经心理功能变化的可能影响
机译:与注意力缺陷多动障碍和COMT val 158 sup> met多态性有关的白质改变:缬氨酸纯合子注意力缺陷多动障碍儿童改变了右扣带(扣带回)的白质连通性
机译:注意力缺陷/多动障碍中MEG背景活动的熵分析
机译:多巴胺转运蛋白基因(DAT1)变异对注意力缺乏/多动障碍脑功能的影响。
机译:去甲肾上腺素转运蛋白基因的多态性改变启动子活性并与注意力缺陷多动障碍相关
机译:去甲肾上腺素转运蛋白基因的多态性改变启动子活性,并与注意力缺陷多动障碍相关