机译:IL-33增强物质P诱导的人肥大细胞分泌VEGF并在牛皮癣皮肤中增加
Molecular Immunopharmacology and Drug Discovery Laboratory, Department of Pharmacology and Experimental Therapeutics, Departments of Department of Obstetrics and Gynecology, Tufts Medical Center, Boston, MA 02111 Biochemistry Department of Obstetrics and Gynecology, Tufts Medical Center, Boston, MA 02111 Internal Medicine, Tufts University School of Medicine, Department of Obstetrics and Gynecology, Tufts Medical Center, Boston, MA 02111 Allergy Clinical Research Center, Division of Allergology, Attikon General Hospital, Athens University Medical School, 12462 Athens, Greecern;
Molecular Immunopharmacology and Drug Discovery Laboratory, Department of Pharmacology and Experimental Therapeutics, Departments of Department of Obstetrics and Gynecology, Tufts Medical Center, Boston, MA 02111 Biochemistry Department of Obstetrics and Gynecology, Tufts Medical Center, Boston, MA 02111rn;
Molecular Immunopharmacology and Drug Discovery Laboratory, Department of Pharmacology and Experimental Therapeutics, Departments of Department of Obstetrics and Gynecology, Tufts Medical Center, Boston, MA 02111rn;
Molecular Immunopharmacology and Drug Discovery Laboratory, Department of Pharmacology and Experimental Therapeutics, Departments of Department of Obstetrics and Gynecology, Tufts Medical Center, Boston, MA 02111 Department of Pharmaceutical Sciences, St. Jude Children's Research Hospital, Memphis, TN 38105Molecular Immunopharmacology and Drug Discovery Laboratory, Department of Pharmacology and Experimental Therapeutics, Departments of Department of Obstetrics and Gynecology, Tufts Medical Center, Boston, MA 02111 Allergy Clinical Research Center, Division of Allergology, Attikon General Hospital, Athens University Medical School, 12462 Athens, Greecern;
Molecular Immunopharmacology and Drug Discovery Laboratory, Department of Pharmacology and Experimental Therapeutics, Departments of Department of Obstetrics and Gynecology, Tufts Medical Center, Boston, MA 02111rn;
Molecular Immunopharmacology and Drug Discovery Laboratory, Department of Pharmacology and Experimental Therapeutics, Departments of Department of Obstetrics and Gynecology, Tufts Medical Center, Boston, MA 02111;
rn;
Molecular Immunopharmacology and Drug Discovery Laboratory, Department of Pharmacology and Experimental Therapeutics, Departments of Department of Obstetrics and Gynecology, Tufts Medical Center, Boston, MA 02111rn;
Allergy Clinical Research Center, Division of Allergology, Attikon General Hospital, Athens University Medical School, 12462 Athens, Greecern;
Molecular Immunopharmacology and Drug Discovery Laboratory, Department of Pharmacology and Experimental Therapeutics, Departments of Department of Obstetrics and Gynecology, Tufts Medical Center, Boston, MA 02111rn;
2nd Department of Dermatology, Attikon General Hospital, Athens University Medical School, 12462 Athens, Greecern;
Maternal-Fetal Medicine, Department of Obstetrics and Gynecology, Tufts Medical Center, Boston, MA 02111rn;
Department of Pharmacology, Boston University Medical School, Boston, MA 02118;
inflammation; cytokines; IL-1; innate immunity; stress;
机译:IL-32在皮损皮损皮肤中与类胰蛋白酶一起增加,并在人肥大细胞中被P物质上调。
机译:IL-33在人体皮肤肥大细胞中的阴阳:降低脱粒,但通过P38激活增强组胺合成
机译:环状AMP通过增强大鼠腮腺腺泡细胞中钙的作用来增强P诱导的淀粉酶分泌
机译:VEGF修饰的人间充质干细胞和脱细胞真皮基质构建组织工程皮肤
机译:肥大细胞和IL-33 / ST2轴是决定碳纳米管毒性的重要因素。
机译:IL-33增强物质P诱导的人类肥大细胞分泌VEGF,并在牛皮癣皮肤中增加
机译:IL-33增强物质P诱导的人类肥大细胞分泌VEGF,并在牛皮癣皮肤中增加