机译:逆转可卡因诱导的突触增强作用,可提供持久保护,防止复发
Departments of Neurosciences, Medical University of South Carolina, Charleston, SC 29425;
Departments of Neurosciences, Medical University of South Carolina, Charleston, SC 29425,Laboratory of Behavioral Neuroscience, Ningbo Addiction Research and Treatment Center, Ningbo University, Ningbo 315000, People's Republic of China;
Departments of Neurosciences, Medical University of South Carolina, Charleston, SC 29425;
Departments of Neurosciences, Medical University of South Carolina, Charleston, SC 29425;
Departments of Neurosciences, Medical University of South Carolina, Charleston, SC 29425;
Departments of Neurosciences, Medical University of South Carolina, Charleston, SC 29425;
Departments of Neurosciences, Medical University of South Carolina, Charleston, SC 29425,Departments of Psychiatry and Behavioral Sciences, Medical University of South Carolina, Charleston, SC 29425;
机译:内源性多巴胺和内胆蛋白信号传导介导可卡因诱导的核心突骨壳中的AMPAR突触潜力的逆转
机译:D2多巴胺受体功能的丧失调节可卡因诱导的腹侧被盖区的谷氨酸能突触增强。
机译:蛋白激酶Mζ是可卡因在腹侧被盖区诱导突触增强所必需的
机译:发射细胞:具有长期突触增强能力的人工神经元。
机译:重获可卡因成瘾:逆转可卡因诱导的可塑性抑制了寻求药物的复发
机译:逆转可卡因诱导的突触增强作用可提供持久保护防止复发
机译:逆转可卡因诱导的突触增强作用,可提供持久保护,防止复发