机译:弓形虫顶膜抗原1的膜内蛋白水解促进宿主细胞的侵袭,但可用于复制
Department of Microbiology and Molecular Genetics, University of Vermont, Burlington, VT 05405;
Department of Microbiology and Molecular Genetics, University of Vermont, Burlington, VT 05405;
Department of Molecular Biology and Genetics, Howard Hughes Medical Institute, The Johns Hopkins University School of Medicine, Baltimore, MD 21205;
Department of Microbiology and Molecular Genetics, University of Vermont, Burlington, VT 05405,Host-Parasite Interactions Section, Laboratory of Intracellular Parasites, Rocky Mountain Laboratories, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Hamilton, MT 59840;
Department of Molecular Biology and Genetics, Howard Hughes Medical Institute, The Johns Hopkins University School of Medicine, Baltimore, MD 21205;
Department of Microbiology and Molecular Genetics, University of Vermont, Burlington, VT 05405;
malaria; regulated intramembrane proteolysis; cell signaling; microneme;
机译:顶膜抗原1是犬新孢子虫和弓形虫之间的交叉反应抗原,抗NcAMA1抗体抑制两种寄生虫入侵宿主细胞。
机译:弓形虫根尖膜抗原1(TgAMA1)的条件表达表明TgAMA1在宿主细胞入侵中起关键作用
机译:弓形虫顶膜抗原-1(TgAMA1)的条件表达表明TgAMA1在宿主细胞侵袭中起关键作用
机译:在开发子宫内膜异位症的女性内膜静脉肌组织和子宫内膜中增加的蛋白质溶解可能促进入侵
机译:功能解析分子机制的主机入侵和复制专心的细胞内病原体弓形虫。
机译:弓形虫顶膜抗原1的膜内蛋白水解促进宿主细胞的侵袭但可用于复制
机译:弓形虫顶膜抗原1的肠内蛋白分解促进了宿主细胞侵袭,但可分配复制
机译:入侵抑制性抗体的作用方式针对恶性疟原虫的顶端膜抗原1