机译:后期极光B的中区激活产生细胞内磷酸化梯度
Departments of Biochemistry and Molecular Genetics, University of Virginia School of Medicine, Charlottesville, Virginia 22908, USA;
Department of Biology, University of Pennsylvania, Philadelphia, Pennsylvania 19104, USA;
Laboratory of Chemistry and Cell Biology, The Rockefeller University, New York, New York 10021, USA;
Departments of Biochemistry and Molecular Genetics, University of Virginia School of Medicine, Charlottesville, Virginia 22908, USA;
Department of Biology, University of Pennsylvania, Philadelphia, Pennsylvania 19104, USA;
Departments of Biochemistry and Molecular Genetics, University of Virginia School of Medicine, Charlottesville, Virginia 22908, USA;
Center for Cell Signaling, University of Virginia School of Medicine, Charlottesville, Virginia 22908, USA;
Departments of Biochemistry and Molecular Genetics, University of Virginia School of Medicine, Charlottesville, Virginia 22908, USA;
Laboratory of Chemistry and Cell Biology, The Rockefeller University, New York, New York 10021, USA;
机译:Aurora A激酶可放大中区磷酸化梯度,以促进高保真细胞分裂。
机译:Cul3-KLHL21 E3泛素连接酶在后期将Aurora B靶向中区微管,是胞质分裂所必需的
机译:Aurora B介导的RASSF1A磷酸化通过将Syntaxin16募集到中区和中体来维持适当的胞质分裂。
机译:各种表面吸附肽和磷酸化抑制剂AG18介导的粘附U937细胞内信号蛋白的LC / MS鉴定
机译:Aurora B介导的有丝分裂和间期磷酸化的作用。
机译:后期Aurora B的中区激活产生细胞内磷酸化梯度。
机译:中源性Auroora B中的半侧激活产生细胞内磷酸化梯度