机译:同种异体IgG结合树突状细胞刺激诱导抗肿瘤T细胞免疫
Stanford Univ, Dept Pathol, Sch Med, Stanford, CA 94305 USA;
Stanford Univ, Dept Pathol, Sch Med, Stanford, CA 94305 USA|Stanford Univ, Sch Med, Dept Microbiol & Immunol, Baxter Lab Stem Cell Biol, Palo Alto, CA 94305 USA;
Stanford Univ, Dept Pathol, Sch Med, Stanford, CA 94305 USA;
Stanford Univ, Sch Med, Dept Cardiothorac Surg, Palo Alto, CA 94305 USA;
Stanford Univ, Dept Pathol, Sch Med, Stanford, CA 94305 USA;
Stanford Univ, Dept Pathol, Sch Med, Stanford, CA 94305 USA;
Stanford Univ, Dept Pathol, Sch Med, Stanford, CA 94305 USA;
Stanford Univ, Dept Pathol, Sch Med, Stanford, CA 94305 USA;
Stanford Univ, Dept Pathol, Sch Med, Stanford, CA 94305 USA;
Stanford Univ, Sch Med, Dept Biochem, Palo Alto, CA 94305 USA;
Stanford Univ, Dept Pathol, Sch Med, Stanford, CA 94305 USA;
Stanford Univ, Dept Pathol, Sch Med, Stanford, CA 94305 USA;
机译:负载有杀死的异源黑色素瘤细胞的树突状细胞可以诱导客观的临床反应和MART-1特异性CD8 + T细胞免疫。
机译:通过在小鼠神经胶质瘤中联合使用替莫唑胺和TAT-survivin脉冲树突状细胞增强抗肿瘤免疫力。
机译:抗CD40诱导的炎症性E-钙黏着蛋白+树突状细胞增强小鼠Lewis肺癌的T细胞反应和抗肿瘤免疫力
机译:通过白细胞uck10-处理的树突细胞和转化生长因子β诱导的调节T细胞抑制人过敏Th2免疫应答的机制,转化生长因子β
机译:组合佐剂诱导的树突细胞交叉呈递和T细胞免疫力的机械洞察
机译:同种异体IgG结合树突状细胞刺激诱导抗肿瘤T细胞免疫
机译:同种异体IgG结合树突细胞刺激诱导抗肿瘤T细胞免疫力