Activation of c-Jun N-terminal kinase 1 and caspase 3 in the tamoxifen-induced apoptosis of rat glioma cells

Sheng-Hong Tseng; Chih-Hsien Wang; Swei-Ming Lin; Chia-Kang Chen; Hsin-Yi Huang; Yun Chen

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Activation of c-Jun N-terminal kinase 1 and caspase 3 in the tamoxifen-induced apoptosis of rat glioma cells

机译：三苯氧胺诱导的大鼠神经胶质瘤细胞凋亡中c-Jun N端激酶1和caspase 3的激活

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The mechanisms of the antitumor effects of tamoxifen upon gliomas are still unclear. In this study, we investigated the role of c-Jun N-terminal kinase-1 (JNK1) and caspase 3 in the tamoxifen-induced apoptosis of rat glioma cells.

机译：他莫昔芬对神经胶质瘤的抗肿瘤作用机制尚不清楚。在这项研究中，我们调查了c-Jun N末端激酶1（JNK1）和caspase 3在他莫昔芬诱导的大鼠神经胶质瘤细胞凋亡中的作用。

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《Journal of Cancer Research and Clinical Oncology》 |2004年第5期|285-293|共9页
作者
Sheng-Hong Tseng; Chih-Hsien Wang; Swei-Ming Lin; Chia-Kang Chen; Hsin-Yi Huang; Yun Chen;
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作者单位

Department of Surgery National Taiwan University Hospital and National Taiwan University College of Medicine;

Department of Surgery National Taiwan University Hospital and National Taiwan University College of Medicine;

Department of Surgery National Taiwan University Hospital and National Taiwan University College of Medicine;

Department of Surgery National Taiwan University Hospital and National Taiwan University College of Medicine;

Department of Surgery National Taiwan University Hospital and National Taiwan University College of Medicine;

Department of Surgery National Taiwan University Hospital and National Taiwan University College of MedicineDepartment of Surgery Far Eastern Memorial Hospital;

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正文语种 eng
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关键词
Apoptosis; Caspase; c-Jun N-terminal kinase; Glioma; Tamoxifen;

机译：凋亡;胱天蛋白酶;c-Jun N-末端激酶;胶质瘤;他莫昔芬;

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1. Activation of c-Jun N-terminal kinase 1 and caspase 3 in the tamoxifen-induced apoptosis of rat glioma cells. [J] . Tseng SH, Wang CH, Lin SM, Journal of Cancer Research and Clinical Oncology . 2004,第5期

机译：他莫昔芬诱导的大鼠神经胶质瘤细胞凋亡中c-Jun N端激酶1和caspase 3的激活。
2. Flavopiridol suppresses tumor necrosis factor-induced activation of activator protein-1, c-Jun N-terminal kinase, p38 mitogen-activated protein kinase (MAPK), p44/p42 MAPK, and Akt, inhibits expression of antiapoptotic gene products, and enhances apoptosis through cytochrome c release and caspase activation in human myeloid cells (Retraction of vol 73, pg 1549, 2008) [J] . Takada Y., Sethi G., Sung B., Molecular pharmacology. . 2016,第1期

机译：黄酮哌啶醇抑制肿瘤坏死因子诱导的活化蛋白1，c-Jun N端激酶，p38丝裂原活化蛋白激酶（MAPK），p44 / p42 MAPK和Akt的激活，抑制抗凋亡基因产物的表达并增强凋亡通过人类髓样细胞中细胞色素c的释放和胱天蛋白酶的活化（Retract of vol 73，pg 1549，2008）
3. Involvement of Asp-Glu-Val-Asp-directed, caspase-mediated mitogen-activated protein kinase kinase 1 Cleavage, c-Jun N-terminal kinase activation, and subsequent Bcl-2 phosphorylation for paclitaxel-induced apoptosis in HL-60 cells. [J] . Shiah SG, Chuang SE, Kuo ML Molecular pharmacology. . 2001,第2期

机译：Asp-Glu-Val-Asp定向，caspase介导的丝裂原激活的蛋白激酶激酶1的参与，c-Jun N端激酶的激活以及随后的Bcl-2磷酸化，用于紫杉醇诱导的HL-60细胞凋亡。
4. Particulate Joint Replacement Materials Induce Apoptosis of Rabbit Synoviocytes Cell Line HIG-82 through c-Jun N-Terminal Kinase (JNK) Pathway [C] . Chu Feng-Min, Lian Shuang-Shii, Sung Yen-Jen International Conference on Biomedical Engineering and Informatics;BMEI '09 . 2009

机译：颗粒关节替代材料通过c-Jun N末端激酶（JNK）途径诱导兔滑膜细胞HIG-82细胞凋亡
5. Role of c-Jun N-terminal kinase (JNK) in mediating mammary cancer cell migration and metastasis. [D] . Mitra, Shreya. 2009

机译：c-Jun N末端激酶（JNK）在介导乳腺癌细胞迁移和转移中的作用。
6. Re: Takada Y Sethi G Sung B and Aggarwal BB (2008) Flavopiridol suppresses tumor necrosis factor-induced activation of activator protein-1 c-Jun N-terminal kinase p38 mitogen-activated protein kinase (MAPK) p44/p42 MAPK and Akt inhibits expression of antiapoptotic gene products and enhances apoptosis through cytochrome c release and caspase activation in human myeloid cells Mol Pharmacol May 2008 73:1549–1557; doi:10.1124/mol.107.041350 [O] . -1

机译：回复：Takada YSethi GSung B和Aggarwal BB（2008）黄酮哌啶醇可抑制肿瘤坏死因子诱导的活化蛋白1c-Jun N端激酶p38丝裂原活化蛋白激酶（MAPK）p44活化/ p42 MAPK和Akt可以抑制抗凋亡基因产物的表达并通过细胞色素c释放和胱天蛋白酶在人骨髓细胞中的活化来增强细胞凋亡Mol Pharmacol2008年5月73：1549-1557； doi：10.1124 / mol.107.041350
7. Re: Takada Y, Sethi G, Sung B, and Aggarwal BB (2008) Flavopiridol suppresses tumor necrosis factor-induced activation of activator protein-1, c-Jun N-terminal kinase, p38 mitogen-activated protein kinase (MAPK), p44/p42 MAPK, and Akt, inhibits expression of antiapoptotic gene products, and enhances apoptosis through cytochrome c release and caspase activation in human myeloid cells Mol Pharmacol May 2008 73:1549–1557; doi:10.1124/mol.107.041350 [O] . 2016

机译：Re：Takada Y，Sethi G，Sung B和Aggarwal BB（2008）抑制肿瘤坏死因子诱导的活化剂蛋白-1，C-6末端激酶，P38丝裂原激活蛋白激酶（MAPK），P44 / p42 mapk和akt抑制抗凋亡基因产物的表达，并通过细胞色素C释放和Caspase活化在人骨髓细胞Mol Pharmacol 2008年5月73：1549-1557中增强细胞凋亡。 DOI：10.1124 / mol.107.041350

Activation of c-Jun N-terminal kinase 1 and caspase 3 in the tamoxifen-induced apoptosis of rat glioma cells

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