首页> 美国卫生研究院文献>Molecular Pharmacology >Re: Takada Y Sethi G Sung B and Aggarwal BB (2008) Flavopiridol suppresses tumor necrosis factor-induced activation of activator protein-1 c-Jun N-terminal kinase p38 mitogen-activated protein kinase (MAPK) p44/p42 MAPK and Akt inhibits expression of antiapoptotic gene products and enhances apoptosis through cytochrome c release and caspase activation in human myeloid cells Mol Pharmacol May 2008 73:1549–1557; doi:10.1124/mol.107.041350

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Re: Takada Y Sethi G Sung B and Aggarwal BB (2008) Flavopiridol suppresses tumor necrosis factor-induced activation of activator protein-1 c-Jun N-terminal kinase p38 mitogen-activated protein kinase (MAPK) p44/p42 MAPK and Akt inhibits expression of antiapoptotic gene products and enhances apoptosis through cytochrome c release and caspase activation in human myeloid cells Mol Pharmacol May 2008 73:1549–1557; doi:10.1124/mol.107.041350

机译：回复：Takada YSethi GSung B和Aggarwal BB（2008）黄酮哌啶醇可抑制肿瘤坏死因子诱导的活化蛋白1c-Jun N端激酶p38丝裂原活化蛋白激酶（MAPK）p44活化/ p42 MAPK和Akt可以抑制抗凋亡基因产物的表达并通过细胞色素c释放和胱天蛋白酶在人骨髓细胞中的活化来增强细胞凋亡Mol Pharmacol2008年5月73：1549-1557； doi：10.1124 / mol.107.041350

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期刊名称 Molecular Pharmacology
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年(卷),期 -1(90),1
年度 -1
页码 63
总页数 1
原文格式 PDF
正文语种
中图分类药学;
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1. Flavopiridol suppresses tumor necrosis factor-induced activation of activator protein-1, c-Jun N-terminal kinase, p38 mitogen-activated protein kinase (MAPK), p44/p42 MAPK, and Akt, inhibits expression of antiapoptotic gene products, and enhances apoptosis through cytochrome c release and caspase activation in human myeloid cells (Retraction of vol 73, pg 1549, 2008) [J] . Takada Y., Sethi G., Sung B., Molecular pharmacology. . 2016,第1期

机译：黄酮哌啶醇抑制肿瘤坏死因子诱导的活化蛋白1，c-Jun N端激酶，p38丝裂原活化蛋白激酶（MAPK），p44 / p42 MAPK和Akt的激活，抑制抗凋亡基因产物的表达并增强凋亡通过人类髓样细胞中细胞色素c的释放和胱天蛋白酶的活化（Retract of vol 73，pg 1549，2008）
2. Flavopiridol suppresses tumor necrosis factor-induced activation of activator protein-1, c-Jun N-terminal kinase, p38 mitogen-activated protein kinase (MAPK), p44/p42 MAPK, and Akt, inhibits expression of antiapoptotic gene products, and enhances apo [J] . Takada Y, Sethi G, Sung B, Molecular pharmacology. . 2008,第5期

机译：黄酮哌啶醇抑制肿瘤坏死因子诱导的活化蛋白1，c-Jun N端激酶，p38丝裂原活化蛋白激酶（MAPK），p44 / p42 MAPK和Akt的激活，抑制抗凋亡基因产物的表达，并增强apo
3. Tumour necrosis factor-induced activation of c-Jun N-terminal kinase is sensitive to caspase-dependent modulation while activation of mitogen-activated protein kinase (MAPK) or p38 MAPK is not [J] . Mohamed AAA., Jupp OJ., Anderson HM., The Biochemical Journal . 2002,第0期

机译：肿瘤坏死因子诱导的c-Jun N末端激酶激活对caspase依赖性调节敏感，而丝裂原激活的蛋白激酶（MAPK）或p38 MAPK激活则不敏感
4. Re: Phromnoi K Reuter S Sung B Prasad S Kannappan R Yadav VR Chanmahasathien W Limtrakul P and Aggarwal BB (2010) A novel pentamethoxyflavone down-regulates tumor cell survival and proliferative and angiogenic gene products through inhibition of IκB kinase activation and sensitizes tumor cells to apoptosis by cytokines and chemotherapeutic agents. Mol Pharmacol 79:279–289; doi:10.1124/mol.110.067512 [O] . -1

机译：回复：Phromnoi KReuter SSung BPrasad SKannappan RYadav VRChanmahasathien WLimtrakul P和Aggarwal BB（2010年）一种新颖的五甲氧基黄酮可通过抑制肿瘤细胞存活以及增殖和血管生成基因产物而下调。 IκB激酶激活并通过细胞因子和化学治疗剂使肿瘤细胞对细胞凋亡敏感。摩尔药学（Mol Pharmacol）79：279–289; doi：10.1124 / mol.110.067512
5. Re: Takada Y, Sethi G, Sung B, and Aggarwal BB (2008) Flavopiridol suppresses tumor necrosis factor-induced activation of activator protein-1, c-Jun N-terminal kinase, p38 mitogen-activated protein kinase (MAPK), p44/p42 MAPK, and Akt, inhibits expression of antiapoptotic gene products, and enhances apoptosis through cytochrome c release and caspase activation in human myeloid cells Mol Pharmacol May 2008 73:1549–1557; doi:10.1124/mol.107.041350 [O] . 2016

机译：Re：Takada Y，Sethi G，Sung B和Aggarwal BB（2008）抑制肿瘤坏死因子诱导的活化剂蛋白-1，C-6末端激酶，P38丝裂原激活蛋白激酶（MAPK），P44 / p42 mapk和akt抑制抗凋亡基因产物的表达，并通过细胞色素C释放和Caspase活化在人骨髓细胞Mol Pharmacol 2008年5月73：1549-1557中增强细胞凋亡。 DOI：10.1124 / mol.107.041350

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