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机译:由于野生型mtDNA分子的丢失,OPA1突变导致细胞色素C氧化酶缺乏
Mitochondrial Research Group, Institute for Ageing and Health, The Medical School, Newcastle University, Newcastle upon Tyne NE2 4HH, UK,|Department of Ophthalmology, Royal Victoria Infirmary, Newcastle upon Tyne, UK,;
Mitochondrial Research Group, Institute for Ageing and Health, The Medical School, Newcastle University, Newcastle upon Tyne NE2 4HH, UK,;
Department of Molecular Physiology and Biophysics, Center for Human Genetics Research, Vanderbilt University Medical Center, Nashville, TN, USA,;
Mitochondrial Research Group, Institute for Ageing and Health, The Medical School, Newcastle University, Newcastle upon Tyne NE2 4HH, UK,|Department of Ophthalmology, Royal Victoria Infirmary, Newcastle upon Tyne, UK,;
Mitochondrial Research Group, Institute for Ageing and Health, The Medical School, Newcastle University, Newcastle upon Tyne NE2 4HH, UK,;
Department of Clinical Medicine, Univer;
机译:由于野生型mtDNA分子的丢失,OPA1突变导致细胞色素C氧化酶缺乏。
机译:mtDNA编码的细胞色素C氧化酶亚基基因中的突变导致孤立的肌病或严重的脑病。
机译:阿尔茨海默氏病患者中细胞色素C氧化酶线粒体DNA中的两个点突变与正常mtDNA共存。
机译:用静脉曲张脱抗乳癌裂缝菌脱氮网的细胞色素C氧化酶的调节
机译:球形球形红球菌的AA3型细胞色素C氧化酶:从D通道中的N139D突变了解质子泵送机制
机译:由于野生型mtDNA分子的丢失OPA1突变导致细胞色素C氧化酶缺乏
机译:由于野生型mtDNA分子的丢失,OPA1突变导致细胞色素C氧化酶缺乏