机译:铁超负荷和糖尿病的风险:遗传性血色素沉着症小鼠模型中从葡萄糖到脂肪酸氧化的转变和肝葡萄糖生产的增加。
Jingyu Huang,1 Deborah Jones,1 Bai Luo,1 Michael Sanderson/ Jamie Soto,1 E. Dale Abel/ Robert C. Cooksey1,2 and Donald A. McClain1'2From the 'Departments of Medicine and Biochemistry, University of Utah School of Medicine, Salt Lake City, Utah, and 2Research Service, VA Medical Center, Salt Lake City, Utah.Corresponding author: Donald A. McClain, donald.mcclain@hsc.utah.edu.Received 26 April 2010 and accepted 20 September 2010. Published ahead of print at httpy7diabetes.diabetesjournals.org on 28 September 2010. DOI: 10.2337/dbl0-0593.© 2011 by the American Diabetes Association. Readers may use this article as long as the work is properly cited, the use is educational and not for profit, and the work is not altered. See http://creativecommons.org/licenses/by -nc-nd/3.0/ for details.The costs of publication of this article were defrayed in part by the payment of page charges. This article must theiefore be hereby marked "advertisement" in accordance with 18 U.S.C. Section 1734 solely to indicate this fact.,;
机译:铁超负荷和糖尿病的风险:遗传性血色素沉着症小鼠模型中从葡萄糖到脂肪酸氧化的转变和肝葡萄糖生产的增加。
机译:在大冢Long-Evans Tokushima肥胖大鼠模型中,在糖尿病前期,增加了肝葡萄糖的产生,并减少了肝葡萄糖的摄取。
机译:在遗传性血色素沉着症患者中,通过静脉穿刺术对铁超负荷标志物正常化后的葡萄糖代谢。
机译:葡萄糖耐受试验过程中葡萄糖和游离脂肪酸动力学的数学模型
机译:胰岛素依赖型糖尿病患者休息和运动期间游离脂肪酸,葡萄糖和胰岛素的动态建模。
机译:铁超负荷和糖尿病的风险:遗传性血色素沉着症小鼠模型中从葡萄糖到脂肪酸氧化的转变和肝葡萄糖生产的增加。
机译:铁超载和糖尿病风险:遗传性血色素沉着症小鼠模型中从葡萄糖到脂肪酸氧化的转变和肝葡萄糖生产的增加。