GIP Contributes to Islet Trihormonal Abnormalities in Type 2 Diabetes

Chee W. Chia; Juliana O. Odetunde; Wook Kim; Olga D. Carlson; Luigi Ferrucci; Josephine M. Egan

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GIP Contributes to Islet Trihormonal Abnormalities in Type 2 Diabetes

机译：GIP有助于2型糖尿病中的胰岛胰岛素异常

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Context:Research and clinical treatments on type 2 diabetes mainly focus on insulin deficiency with little attention paid to other islet hormones.

机译：背景：2型糖尿病的研究和临床治疗主要关注胰岛素缺乏，几乎没有注意其他胰岛激素。

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《The journal of clinical endocrinology and metabolism》 |2014年第7期|共9页
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Chee W. Chia; Juliana O. Odetunde; Wook Kim; Olga D. Carlson; Luigi Ferrucci; Josephine M. Egan;
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1. Abnormally decreased NO and augmented CO production in islets of the leptin-deficient ob/ob mouse might contribute to explain hyperinsulinemia and islet survival in leptin-resistant type 2 obese diabetes. [J] . Jimenez Feltstrom J, Salehi A, Meidute Abaraviciene S, Regulatory peptides. . 2011,第1a3期

机译：瘦素缺陷型ob / ob小鼠的胰岛中NO异常减少和CO产生增加可能有助于解释高胰岛素血症和瘦素抵抗性2型肥胖糖尿病患者的胰岛存活。
2. Chemical Ablation of Gastric Inhibitory Polypeptide Receptor Action by Daily (Pro3)GIP Administration Improves Glucose Tolerance and Ameliorates Insulin Resistance and Abnormalities of Islet Structure in Obesity-Related Diabetes. [J] . Gault VA, Irwin N, Green BD, Diabetes . 2005,第8期

机译：通过每日（Pro3）GIP给药化学抑制胃抑制性多肽受体的作用可改善肥胖相关糖尿病患者的糖耐量，改善胰岛素抵抗和胰岛结构异常。
3. Development of a diet-induced murine model of diabetes featuring cardinal metabolic and pathophysiological abnormalities of type 2 diabetes Development of a diet-induced murine model of diabetes featuring cardinal metabolic and pathophysiological abnormalities of type 2 diabetes Development of a diet-induced murine model of diabetes featuring cardinal metabolic and pathophysiological abnormalities of type 2 diabetes [J] . Natkunam Ketheesan, Tahnee L. Bridson, Brenda L. Govan, Biology Open . 2016,第8期

机译：以2型糖尿病的主要代谢和病理生理异常为特征的饮食诱导型糖尿病鼠模型的开发以2型糖尿病的主要的代谢和病理生理异常为特征的饮食诱导的糖尿病鼠模型的开发表现为2型糖尿病的主要代谢和病理生理异常
4. Frequency-Based Releasing of Insulin in the Langerhans Islets To Counteract the Beginning of the Type-2 Diabetes in Pre-diabetes Patients [C] . Huber Nieto-Chaupis IEEE Annual Computing and Communication Workshop and Conference . 2019

机译：郎格罕岛胰岛中基于频率的胰岛素释放以抵消糖尿病前期患者中2型糖尿病的发作
5. Gaz: An Intra-islet Signaling Hub Fundamental for Type 1 Diabetes Pathophysiology [D] . Fenske, Rachel Joanne 2019

机译：Gaz：1型糖尿病病理生理学的胰岛内信号传导枢纽基础
6. GIP Contributes to Islet Trihormonal Abnormalities in Type 2 Diabetes [O] . Chee W. Chia, Juliana O. Odetunde, Wook Kim, -1

机译：GIP导致2型糖尿病的胰岛三体激素异常
7. Chemical ablation of gastric inhibitory polypeptide receptor action by daily (Pro3)GIP administration improves glucose tolerance and ameliorates insulin resistance and abnormalities of islet structure in obesity-related diabetes. [O] . Gault, Victor A, Irwin, Nigel, Green, Brian D, 2005

机译：每天（Pro3）GIP给药对胃抑制性多肽受体作用的化学消融可改善肥胖相关糖尿病患者的糖耐量，改善胰岛素抵抗和胰岛结构异常。

GIP Contributes to Islet Trihormonal Abnormalities in Type 2 Diabetes

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