Transcription Factor KLLN Inhibits Tumor Growth by AR Suppression, Induces Apoptosis by TP53/TP73 Stimulation in Prostate Carcinomas, and Correlates With Cellular Differentiation

Yu Wang; Deepa Radhakrishnan; Xin He; Donna M. Peehl; Charis Eng

首页> 外文期刊>The journal of clinical endocrinology and metabolism >Transcription Factor KLLN Inhibits Tumor Growth by AR Suppression, Induces Apoptosis by TP53/TP73 Stimulation in Prostate Carcinomas, and Correlates With Cellular Differentiation

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Transcription Factor KLLN Inhibits Tumor Growth by AR Suppression, Induces Apoptosis by TP53/TP73 Stimulation in Prostate Carcinomas, and Correlates With Cellular Differentiation

机译：转录因子KLLN抑制AR抑制肿瘤生长，通过TP53 / TP73刺激诱导前列腺癌细胞凋亡，并与细胞分化相关。

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Context: KLLN is a newly identified gene with unknown function and shares a bidirectional promoter with PTEN .

机译：背景：KLLN是一个新近鉴定的功能未知的基因，与PTEN共享一个双向启动子。

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《The journal of clinical endocrinology and metabolism》 |2013年第3期|共9页
作者
Yu Wang; Deepa Radhakrishnan; Xin He; Donna M. Peehl; Charis Eng;
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1. Androgen receptor-induced tumor suppressor, KLLN, inhibits breast cancer growth and transcriptionally activates p53/p73-mediated: Apoptosis in breast carcinomas [J] . WangY., HeX., YuQ., Human Molecular Genetics . 2013,第11期

机译：雄激素受体诱导的肿瘤抑制因子KLLN抑制乳腺癌的生长并转录激活p53 / p73介导的乳腺癌细胞凋亡
2. KLF9, a transcription factor induced in flutamide-caused cell apoptosis, inhibits AKT activation and suppresses tumor growth of prostate cancer cells [J] . ShenP., SunJ., XuG., The Prostate . 2014,第9期

机译：KLF9是在氟他胺引起的细胞凋亡中诱导的转录因子，可抑制AKT活化并抑制前列腺癌细胞的肿瘤生长
3. Novel C-4 Heteroaryl 13-cis-Retinamide Mnk/AR Degrading Agents Inhibit Cell Proliferation and Migration and Induce Apoptosis in Human Breast and Prostate Cancer Cells and Suppress Growth of MDA-MB-231 Human Breast and CWR22Rv1 Human Prostate Tumor Xenografts in Mice [J] . Mbatia Hannah W., Ramalingam Senthilmurugan, Ramamurthy Vidya P., Journal of Medicinal Chemistry . 2015,第4期

机译：新型C-4杂芳基13-顺式视黄酰胺Mnk / AR降解剂抑制人乳腺癌和前列腺癌细胞的增殖和迁移并诱导其凋亡，并抑制MDA-MB-231人乳腺癌和CWR22Rv1人前列腺肿瘤异种移植物的生长。
4. Total Paeony Glycoside Inhibits Tumor Growth and Induces Apoptosis in K562 Cells and in S180 Tumor-Bearing Mice [C] . Huiyu Xu, Yanmin Wu, Xiaoqing Luo, Information technology and agricultural engineering . 2011

机译：总Pa药苷抑制K562细胞和S180荷瘤小鼠的肿瘤生长并诱导其凋亡。
5. The role of p38 mitogen activated protein kinase in post-transcriptional regulation of cyclooxygenase-2 in intleukin-1beta and tumor necrosis factor-alpha stimulated prostate cancer cell lines. [D] . Khatri, Jitender. 2011

机译：p38丝裂原活化蛋白激酶在intleukin-1beta和肿瘤坏死因子-α刺激的前列腺癌细胞系中环氧合酶2的转录后调控中的作用。
6. Transcription Factor KLLN Inhibits Tumor Growth by AR Suppression Induces Apoptosis by TP53/TP73 Stimulation in Prostate Carcinomas and Correlates With Cellular Differentiation [O] . Yu Wang, Deepa Radhakrishnan, Xin He, -1

机译：转录因子KLLN抑制AR抑制肿瘤生长通过TP53 / TP73刺激诱导前列腺癌细胞凋亡并与细胞分化相关
7. Transcription Factor KLLN Inhibits Tumor Growth byARSuppression, Induces Apoptosis byTP53/TP73Stimulation in Prostate Carcinomas, and Correlates With Cellular Differentiation [O] . Yu Wang, Deepa Radhakrishnan, Xin He, 2013

机译：转录因子Klln抑制肿瘤生长Byarsup抑制，诱导前列腺癌中的细胞凋亡，诱导细胞瘤癌，并与细胞分化相关
8. Identification of Sonic Hedgehog-Induced Stromal Factors That Stimulate Prostate Tumor Growth; Annual summary rept. 1 Nov 2006-31 Oct 2007 [R] . Bushman, W., Shaw, A. 2007

机译：sonic Hedgehog诱导的促进前列腺肿瘤生长的基质因子的鉴定;年度总结报告2006年11月1日至2007年10月31日

Transcription Factor KLLN Inhibits Tumor Growth by AR Suppression, Induces Apoptosis by TP53/TP73 Stimulation in Prostate Carcinomas, and Correlates With Cellular Differentiation

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