The aipokine zinc-α2-glycoprotein is down regulated with fat mass expansion in obesity

C. Bing; J. Pinkney; J. Wilding; K. Kos; P. Trayhurn; Q. Ding; T. Mracek; T. Tzanavari

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The aipokine zinc-α2-glycoprotein is down regulated with fat mass expansion in obesity

机译：肥胖中脂肪含量的增加会降低脂肪蛋白锌-α2-糖蛋白的表达

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《Proceedings of the Nutrition Society 》 |2009年第oce2期| 共页
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C. Bing; J. Pinkney; J. Wilding; K. Kos; P. Trayhurn; Q. Ding; T. Mracek; T. Tzanavari;
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中图分类营养学 ;
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1. Zinc-α2-glycoprotein 1 attenuates non-alcoholic fatty liver disease by negatively regulating tumour necrosis factor-α [J] . Ting Liu, Xin Luo, Zheng-Hong Li, World Journal of Gastroenterology . 2019 ,第36期

机译：锌-α2-糖蛋白1通过负调节肿瘤坏死因子-α衰减非酒精性脂肪肝疾病
2. Zinc-|[alpha]|2-glycoprotein: an adipokine modulator of body fat mass|[quest]| [J] . C Bing, T Mracek, D Gao, International Journal of Obesity . 2010 ,第11期

机译：锌-|α| 2-糖蛋白：体脂质量的脂肪因子调节剂|
3. Depot- and obesity-related differences in adipogenesisAdipocyte hypertrophy and hyperplasia are known to facilitate lipid storage in adipose tissues by increasing adipocyte cell size and number, respectively. Adipogenesis is the process resulting in adipose tissue hyperplasia. Although depot-specific differences and obesity-related modulation of adipocyte size are well documented, available data on adipogenesis and adipose tissue hyperplasia are less conclusive. Most studies support a reduction of adipogenesis in the obese state. Preadipocytes of the subcutaneous fat depot appear to be more responsive to adipogenic stimulation compared with those from visceral fat compartments in most studies. A number of studies support the notion that adipose tissue expansion through hyperplasia reduces ectopic lipid excess and obesity-related complications. Several genetic variants have been identified in the genes coding for adipogenesis-regulating proteins. While some of these variants have been clearly associated with the phenotypes of obesity and obesity-related alterations, available data highlight the importance of considering gene–gene and gene–diet interactions. [J] . Julie. Lessard, André. Tchernof Clinical lipidology. . 2012 ,第5期

机译：脂肪形成与肥胖相关的差异已知脂肪细胞肥大和增生分别通过增加脂肪细胞的大小和数量来促进脂质在脂肪组织中的存储。脂肪形成是导致脂肪组织增生的过程。尽管已经有很多文献记载了贮库特异性差异和肥胖相关的脂肪细胞大小调节，但有关脂肪形成和脂肪组织增生的可用数据尚无定论。大多数研究支持在肥胖状态下减少脂肪形成。在大多数研究中，与来自内脏脂肪区室的脂肪细胞相比，皮下脂肪库的前脂肪细胞似乎对脂肪刺激更为敏感。许多研究支持这样的观点，即通过增生的脂肪组织扩张可以减少异位脂质过多和肥胖相关的并发症。在编码脂肪形成调节蛋白的基因中已经鉴定出几种遗传变异。尽管其中一些变异与肥胖症的表型和与肥胖有关的改变明显相关，但现有数据突出了考虑基因-基因和基因-饮食相互作用的重要性。
4. Self-Regulated Learning Differences between Obese and Non-Obese Adolescents: A Comparative Study of Senior High School Students in Medan City [C] . Fasti Rola, Ade Rahmawati Srg, Debby A. Daulay International Conference on Social and Political Development . 2018

机译：肥胖与非肥胖青少年之间的自我监管学习差异：棉兰城市高中生的比较研究
5. Lymphotoxin and high fat diet dynamically regulate the microbiota to enable obesity. [D] . Upadhyay, Vaibhav. 2013

机译：淋巴毒素和高脂饮食可以动态调节微生物群，从而实现肥胖。
6. Novel Aspects of Adipocyte Biology: High-fat diet-induced obesity regulates MMP3 to modulate depot- and sex-dependent adipose expansion in C57BL/6J mice [O] . Yuanyuan Wu, Mi-Jeong Lee, Yasuo Ido, -1

机译：脂肪细胞生物学的新方面：高脂饮食诱导的肥胖症调节MMP3来调节C57BL / 6J小鼠的贮库和性别依赖性脂肪扩张
7. Circulating and Adipose Tissue Gene Expression of Zinc-α2-Glycoprotein in Obesity: Its Relationship with Adipokine and Lipolytic Gene Markers in Subcutaneous and Visceral Fat [O] . V. Ceperuelo-Mallafré, S. Näf, X. Escoté, 2009

机译：锌-α2-糖蛋白在肥胖症中的循环和脂肪组织基因表达：皮下和内脏脂肪中脂质和脂溶性基因标志物的关系

The aipokine zinc-α2-glycoprotein is down regulated with fat mass expansion in obesity

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