Proteasome inhibition and stress compromise the heart in chemotherapy

Yolande E.A. Appelman1 and Pieter A. Doevendans2*

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Proteasome inhibition and stress compromise the heart in chemotherapy

机译：蛋白酶体抑制和压力在化疗中损害心脏

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来源
《Cardiovascular Research》 |2008年第4期|p.547-548|共2页
作者
Yolande E.A. Appelman1 and Pieter A. Doevendans2*;
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1Department of Cardiology, VU University Medical Center, Amsterdam, The Netherlands 2Department of Cardiology, University Medical Center Utrecht, PO Box 85500, GA Utrecht 3508, The Netherlands;

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1. Proteasome inhibition and stress compromise the heart in chemotherapy [J] . Yolande E.A. Appelman, Pieter A. Doevendans Cardiovascular Research . 2008,第4期

机译：蛋白酶体抑制和压力在化疗中损害心脏
2. Too much of a good thing is bad: proteasome inhibition induces stressed hearts to fail. [J] . Carrier L Cardiovascular Research . 2010,第3期

机译：太多的好事是坏的：蛋白酶体的抑制会引起压力大的心脏衰竭。
3. Too much of a good thing is bad: proteasome inhibition induces stressed hearts to fail [J] . Lucie Carrier Cardiovascular Research . 2010,第3期

机译：太多的好事是坏的：蛋白酶体抑制会导致压力大的心脏衰竭
4. Arsenic trioxide induces both apoptosis and autophagy through stimulation of ER stress and inhibition of ubiquitin-proteasome system in human sarcoma cells [C] . H.-W. Chiu, Y.-C. Tseng, Y.-J. Wang International Congress on Arsenic in the Environment . 2014

机译：通过刺激人类肉瘤细胞中的ER应激和泛素 - 蛋白酶体系的ER应激和抑制来诱导细胞凋亡和自噬
5. Pharmacodynamic systems analysis of HDAC and proteasome inhibition in multiple myeloma. [D] . Nanavati, Charvi. 2016

机译：多发性骨髓瘤中HDAC和蛋白酶体抑制的药效系统分析。
6. Autophagy Inhibition Compromises Degradation of Ubiquitin-Proteasome Pathway Substrates [O] . Viktor I. Korolchuk, Alicia Mansilla, Fiona M. Menzies, -1

机译：自噬抑制损害泛素-蛋白酶体途径底物的降解。
7. Proteasome inhibition and stress compromise the heart in chemotherapy [O] . Y. E.A. Appelman, P. A. Doevendans 2008

机译：蛋白酶体抑制和应力抑制了化疗中的心脏

Proteasome inhibition and stress compromise the heart in chemotherapy

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