机译:BAG-1在结直肠肿瘤进展中上调,并通过增加NF-κB活性促进结直肠肿瘤细胞存活
Cancer Research UK Colorectal Tumour Biology Research Group, Department of Cellular and Molecular Medicine, University of Bristol, Bristol BS8 1TD, UK 1Institute of Molecular Plant Sciences, University of Edinburgh EH9 3JH, Edinburgh, UK 2Department of Pathology and Histology, Bristol Royal Infirmary, Bristol BS2 8HW, UK 3Cancer Sciences Division, Cancer Research UK Clinical Centre, University of Southampton SO16 6YD, UK 4Department of Oral and Dental Science, University of Bristol, Bristol BS1 2LY, UK;
机译:BAG-1在结直肠肿瘤进展中上调,并通过增加NF-κB活性促进结直肠肿瘤细胞存活。
机译:上调的miR-17通过靶向RND3抑癌基因在大肠癌中促进细胞增殖,肿瘤生长和细胞周期进程
机译:上调的miR-17通过靶向RND3抑癌基因在大肠癌中促进细胞增殖,肿瘤生长和细胞周期进程
机译:使用MALDI质谱成像的人结直肠癌肿瘤微环境相互作用的代谢特征
机译:捕获大肠循环肿瘤细胞以进行突变分析
机译:原始文章:通过基因扩增增加Solute载体家族12成员5的表达有助于肿瘤进展和转移并与大肠癌的不良生存有关
机译:BAG-1在结肠直肠肿瘤进展中上调,通过增加NF-κB活性来促进结肠直肠肿瘤细胞存活