closure for a press for injection moulding: a duese of massezylinders for plastic masses

• 1. 具有齿圈缸和顶件缸的精冲压力机 [P] . 中国专利： CN1219895A . 1999-06-16
• 2. 家具用塑料嵌件移动式压注模 [P] . 中国专利： CN205326104U . 2016.06.22
• 3. closure for a press for injection moulding: a duesenoeffnung of massezylinders [P] . 德国专利： DE000000661579A . 1938-06-22

  机译： 注塑机压力机的闭合：喷砂缸的双重作用

• 4. closure for a press for injection moulding: a spritzduese for plastic masses [P] . 德国专利： DE000000712182A . 1941-10-14

  机译： 用于注塑机的封闭件：用于塑料块的喷头

• 5. The form for the production of shaped articles made of plastic masses, in particular curable plastics, in the hot - or injection moulding press the method under the press [P] . 德国专利： DE1479796A1 . 1969-06-19

  机译： 在热压或注塑成型压力机中生产由塑料块，特别是可固化塑料制成的成型制品的形式

• 1. Haitian Plastic Injection Moulding Machines [J] . 中国对外贸易：英文版 . 1999,第003期
• 2. Effect of intramuscular calcitonin injection combined with intra-articular ozone injection on the expression of the IL family and MMPs family of knee osteoarthritis [J] . Liang Luo 海南医科大学学报（英文版） . 2017,第019期
• 3. Effect of intramuscular calcitonin injection combined with intra-articular ozone injection on the expression of the IL family and MMPs family of knee osteoarthritis [J] . Liang Luo 海南医科大学学报(英文版) . 2017,第019期
• 4. The Effect of Pressure on Warpage of Dumbbell Plastic Part in Injection Moulding Machine [C] . Mohd Amran, Siti Salmah, Mohd Zaki International Manufacturing Engineering Conference . 2014
• 5. Development of an injection and moulding subsystem for a microcellular plastic manufacturing process. [D] . Ozolins, Christopher. 2001
• 6. Development and Characterization of Environmentally Friendly Wood Plastic Composites from Biobased Polyethylene and Short Natural Fibers Processed by Injection Moulding [O] . Celia Dolza, Eduardo Fages, Eloi Gonga, 2021
• 7. Injection and injection-compression moulding replication capability for the production of polymer lab-on-a-chip with nano structures [O] . M Calaon, G Tosello, J Garnaes, 2017
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• 1. Study on Injection Moulding of High Precision Aspheric Plastic Lens [J] . TO ,Suet ,LEE . 厦门大学学报：自然科学版 . 2002,第0S1期
• 2. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004） （P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期
• 3. 消失模铸造质量管理泡沫塑料模样的首件必检制度 [C] . 袁东洲 . 2014年第二届西部铸造论坛 . 2014
• 4. 基于多软件、多平台的塑料模标准件系统的研究与开发 [A] . 马万杰 . 2009