method to reduce the ash content of olefinischen polymers and copolymers

• 1. 除去乙烯-α烯烃共聚物聚合物溶液中的催化剂的方法及共聚物的制备方法 [P] . 中国专利： CN103374089B . 2015.10.07
• 2. 除去乙烯-α烯烃共聚物聚合物溶液中的催化剂的方法及共聚物的制备方法 [P] . 中国专利： CN103374089A . 2013-10-30
• 3. Method for reducing the ash content of olefin polymers and copolymers [P] . 英国专利： GB960335A . 1964-06-10

  机译： 降低烯烃聚合物和共聚物的灰分含量的方法

• 4. Improved process for making it possible to reduce the ash content of the polymers and copolymers of alkylene oxides [P] . 法国专利： FR1543934A . 1968-10-31

  机译： 改进的方法，可以降低环氧烷烃的聚合物和共聚物的灰分含量

• 5. Process for reducing the ash content of the olefinic polymers and copolymers [P] . 法国专利： FR1345830A . 1963-12-13

  机译： 降低烯烃聚合物和共聚物的灰分含量的方法

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• 7. Block Copolymer Synthesis by the Combination of Living Cationic Polymerization and Other Polymerization Methods [O] . Asmita Dey, Ujjal Haldar, Priyadarsi De 2021
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• 1. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004） （P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期
• 2. Morphology Control of Polymer Microspheres Containing Block Copolymers with Seed Polymerization [J] . Hossein Taherzadeh ,Shunsuke Sotowa ,Kenji Ogino . 有机高分子材料期刊（英文） . 2015,第2期
• 3. 合成环化双烯烃聚合物及共聚物的若干新进展 [C] . 杨继华 ,逄束芬 ,董为民 . 2001年全国高分子学术论文报告会 . 2001
• 4. 聚合物添加剂对环烯烃共聚物芯片电泳分析性能的影响及其应用 [A] . 李红丽 . 2020