method and device for center of mass along its schaftachse marked with unequal rotationskörpern

• 1. 不规则物体质心测量方法 [P] . 中国专利： CN111024309A . 2020-04-17
• 2. 基于3D扫描仪的不规则物体质心的测量装置及方法 [P] . 中国专利： CN107782499B . 2019.12.24
• 3. Method and apparatus for centering of with an unequal distribution of mass is provided along its axis of the shaft rotation bodies [P] . 德国专利： DE2823219A1 . 1979-11-29

  机译： 沿着轴旋转体的轴线设置有用于以不均匀的质量分布对中的方法和设备

• 4. method and device for determining the unwuchterzwungenen wellenschwingungen of horizontally mounted rotationskörpern [P] . 德国专利： DE3044440C2 . 1991-09-19

  机译： 确定水平安装的旋转的不稳定性的方法和装置

• 5. method and device for the compensation of unwuchten in at least two layers of rotationskörpern, the units for unbalance correction only in constructive set ausgleichsebenen and limited winkelbereichen [P] . 德国专利： DE3102963C2 . 1986-07-03

  机译： 于至少两层旋转中的不平衡补偿的方法和装置，仅用于建设性设置的有限元法和有限的winkelbereichen中的不平衡校正单元

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• 5. The New Food and Drug Administration Center for Devices and Radiological Health Engineering and Physics Laboratory and Methods for Testing Software That Controls Infusion Pumps [O] . David C. Klonoff 2007
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• 1. Feedback-based unequal error protection fountain code over device-to-device broadcast [J] . 黄盖世 ,费泽松 ,曹聪哲 . 北京理工大学学报：英文版 . 2014,第4期
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• 3. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004） （P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期
• 4. 角度校准中不规则旋转体同轴调整方法 [C] . LI Qian ,李倩 ,SUN An-bin . 2012年全国几何量精密测量技术学术交流会 . 2012
• 5. 基于加权质心矿井移动设备定位方法的研究 [A] . 金曼曼 . 2018