Changes in CNS Metabolism in High Pressure Nervous Syndrome, High Pressure Narcosis, and Methods of Protection.

摘要

The authors initial approach to the study of mechanisms of acute central nervous system high pressure toxicity was to study enzyme activities and concentrations of the series of metabolites (which have been shown to provide protection against O2 toxicity) during air and hyperbaric oxygen exposures - 5 ATA O2 - 11 ATA O2, with or without exogenous administration of the particular protective agent.