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首页> 外文期刊>Movement disorders >Etiological musculo-skeletal factor in focal dystonia in a musician's hand: A case study of the right hand of a guitarist.
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Etiological musculo-skeletal factor in focal dystonia in a musician's hand: A case study of the right hand of a guitarist.

机译:音乐家手中局灶性肌张力障碍的病因肌肉骨骼因素:以吉他手的右手为例。

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摘要

A case study is presented in which a focal hand dystonia seems to have developed in the right hand of a classical guitarist as a result of a neuromuscular peripheral defect caused by trauma. The trauma was a near total perforation of the first web space by a splinter. Healing was uneventful without apparent functional complications. Two years later the patient noticed difficulties in extending the index in playing, for which he received various unsuccessful treatments during seven years. However, we found more severe dystonic symptoms (cocontractions) in the thumb than in the index during playing, which correlated with an undiagnosed insufficiency in the flexor pollicis brevis (FPB). This defect allowed proposing a biomechanical analysis of compensations for diminished thumb control in playing, which would explain the dysfunction in the index in playing as overcompensation for the thumb problem. If this analysis is correct, the etiology of the case can be traced back to underlying multiarticular control problems in the thumb caused by an insufficient FPB. This defect was considered irrepairable. It was concluded that even with knowledge of the underlying cause, a potentially successful treatment of the dystonia might not exist in this case. The case would demonstrate that task-specific hand dystonias can arise as overcompensations for (peripheral) neuro-musculoskeletal defects. The case is illustrated by videos of playing and functional thumb tests.
机译:提出了一个案例研究,其中由于创伤引起的神经肌肉周围缺损,古典吉他手的右手似乎出现了局灶性手张力障碍。创伤是第一个网状空间几乎被碎片刺穿。愈合没有任何明显的功能并发症。两年后,患者注意到难以扩大演奏指数,因此他在七年内接受了各种治疗失败。但是,我们发现在玩耍过程中,拇指的肌张力障碍症状(共收缩)比食指更严重,这与短屈肌弯曲症(FPB)的未确诊不足有关。该缺陷允许提出对演奏中拇指控制减弱的补偿进行生物力学分析的方法,这将解释演奏索引中的功能障碍是对拇指问题的过度补偿。如果此分析是正确的,则该病例的病因可以追溯到由FPB不足引起的拇指潜在的多关节控制问题。该缺陷被认为是无法修复的。结论是,即使了解潜在的原因,这种情况下也不可能成功治疗肌张力障碍。该案例表明,针对(周围)神经肌肉骨骼缺陷的过度补偿可能导致特定任务的手肌张力障碍。通过播放视频和功能拇指测试来说明该案例。

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