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Fracture and contralateral dislocation of the twin facet joints of the lower cervical spine.

机译:下颈椎双小关节的骨折和对侧脱位。

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PURPOSE: The combination of a facet fracture and a contralateral facet dislocation at the same intervertebral level of the cervical spine (a fracture and contralateral dislocation of the twin facet joints) has not been described in detail. The aims of this study are to report a series of 11 patients with this injury, to clarify the clinical features and to discuss its pathomechanism. METHODS: Among 251 patients with lower cervical spine fractures and/or dislocations surgically treated, 11 (9 males and 2 females, averaged age, 52 years) had this kind of injury. Medical charts and medical images were reviewed retrospectively. RESULTS: Injury levels were C4-5, C5-6 and C6-7 in 1, 4 and 6 patients, respectively. A fracture was found at the superior facet in 6, and at the inferior facet in 5. The anterior displacement of the vertebral body ranged from 7 to 19 mm. The unilateral horizontal facet appearance on an anteroposterior radiograph and the triple image on a CT composed of a separated fracture fragment, the base of the fractured facet, and the neighboring non-fractured facet were characteristic. All patients had neurological deficits from Frankel A to D, and were surgically treated by posterior fusion using wire or cable, or combined anterior and posterior spinal fusion. CONCLUSIONS: The fracture and contralateral dislocation of the twin facet joints can cause severe neurological deficits because of its gross anterior displacement. Its plausible pathomechanism is extension force exerted to the cervical spine when it is maximally bent laterally.
机译:目的:没有详细描述在同一颈椎水平上小平面骨折和对侧小关节脱位(双小关节的骨折和对侧脱位)的组合。这项研究的目的是报告一系列11例这种损伤的患者,以阐明其临床特征并讨论其致病机理。方法:通过手术治疗的251例下颈椎骨折和/或脱位患者中,有11例(男性9例,女性2例,平均年龄52岁)受到了这种伤害。回顾性检查病历和医学图像。结果:1、4和6例患者的损伤水平分别为C4-5,C5-6和C6-7。在6的上小面发现骨折,在5的下小面发现骨折。椎体的前移范围为7到19 mm。前后位X线片的单侧水平面外观和CT图像由分离的骨折片段,骨折面的基部和相邻的未破裂面组成。从弗兰克尔(Frankel)A到D,所有患者均存在神经功能缺损,并通过钢丝或电缆的后路融合术或前路和后路脊柱融合术进行手术治疗。结论:双小关节的骨折和对侧脱位可导致严重的神经功能缺损,原因是其前位移位。其合理的病理机制是当最大程度地侧向弯曲时施加到颈椎的伸展力。

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