机译:LMO2通过脱乙酰化活化对于血液缺陷和T-all白血病是必不可少的
Univ Hosp Tubingen Dept Hematol Oncol Clin Immunol &
Rheumatol Otfried Mueller Str 10 D-72076;
Univ Hosp Tubingen Dept Hematol Oncol Clin Immunol &
Rheumatol Otfried Mueller Str 10 D-72076;
Univ Hosp Tubingen Dept Hematol Oncol Clin Immunol &
Rheumatol Otfried Mueller Str 10 D-72076;
Univ Hosp Tubingen Dept Hematol Oncol Clin Immunol &
Rheumatol Otfried Mueller Str 10 D-72076;
Univ Hosp Tubingen Dept Hematol Oncol Clin Immunol &
Rheumatol Otfried Mueller Str 10 D-72076;
Univ Hosp Tubingen Dept Hematol Oncol Clin Immunol &
Rheumatol Otfried Mueller Str 10 D-72076;
Univ Hosp Tubingen Dept Hematol Oncol Clin Immunol &
Rheumatol Otfried Mueller Str 10 D-72076;
Univ Hosp Tubingen Dept Hematol Oncol Clin Immunol &
Rheumatol Otfried Mueller Str 10 D-72076;
Max Planck Inst Dev Biol Dept Prot Evolut Tubingen Germany;
German Ctr Neurodegenerat Dis Tubingen Germany;
Univ Hosp Tubingen Dept Hematol Oncol Clin Immunol &
Rheumatol Otfried Mueller Str 10 D-72076;
Univ Hosp Tubingen Dept Hematol Oncol Clin Immunol &
Rheumatol Otfried Mueller Str 10 D-72076;
Univ Hosp Tubingen Dept Hematol Oncol Clin Immunol &
Rheumatol Otfried Mueller Str 10 D-72076;
Univ Hosp Tubingen Dept Hematol Oncol Clin Immunol &
Rheumatol Otfried Mueller Str 10 D-72076;
Univ Hosp Tubingen Dept Hematol Oncol Clin Immunol &
Rheumatol Otfried Mueller Str 10 D-72076;
Univ Hosp Tubingen Dept Hematol Oncol Clin Immunol &
Rheumatol Otfried Mueller Str 10 D-72076;
Univ Childrens Hosp Tubingen Tubingen Germany;
Univ Childrens Hosp Tubingen Tubingen Germany;
Univ Hosp Tubingen Inst Med Genet &
Appl Genom Tubingen Germany;
Univ Childrens Hosp Tubingen Tubingen Germany;
Univ Childrens Hosp Tubingen Tubingen Germany;
Univ Hosp Tubingen Dept Hematol Oncol Clin Immunol &
Rheumatol Otfried Mueller Str 10 D-72076;
Univ Hosp Tubingen Dept Hematol Oncol Clin Immunol &
Rheumatol Otfried Mueller Str 10 D-72076;
Univ Hosp Tubingen Dept Hematol Oncol Clin Immunol &
Rheumatol Otfried Mueller Str 10 D-72076;
机译:LMO2通过脱乙酰化活化对于血液缺陷和T-all白血病是必不可少的
机译:Nampt / Sirt2介导的LMO2通过脱乙酰化活化对于血缺陷是必不可少的
机译:通过组织上获得的新立方促进剂激活T-全部的LMO2癌基因
机译:小鼠胚胎中造血功能和血管生成的激活:原始内胚层的信号诱导和重编程外胚层细胞的命运
机译:Tafazzin在血缺陷和白血病的作用
机译:Notch1抑制作用靶向T-ALL的Tal1 / Lmo2小鼠模型中的白血病起始细胞
机译:Notch1抑制靶向T-aLL的Tal1 / Lmo2小鼠模型中的白血病起始细胞