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机译:COX-2和5-LOX对COX-2和5-LOX的抑制通过促进PTEN和抑制PI3K / AKT途径来调节结肠直肠癌的进展
Wuhan Univ Renmin Hosp Dept Pancreat Surg 99 Zhangzhidong Rd Wuhan 430030 Hubei Peoples R;
Huazhong Univ Sci &
Technol Union Hosp Dept Neurosurg Tongji Med Coll Wuhan Hubei Peoples R;
Wuhan Univ Renmin Hosp Dept Pancreat Surg 99 Zhangzhidong Rd Wuhan 430030 Hubei Peoples R;
Wuhan Univ Renmin Hosp Dept Pancreat Surg 99 Zhangzhidong Rd Wuhan 430030 Hubei Peoples R;
Wuhan Univ Renmin Hosp Dept Pancreat Surg 99 Zhangzhidong Rd Wuhan 430030 Hubei Peoples R;
Wuhan Univ Renmin Hosp Dept Pancreat Surg 99 Zhangzhidong Rd Wuhan 430030 Hubei Peoples R;
Wuhan Univ Renmin Hosp Dept Pancreat Surg 99 Zhangzhidong Rd Wuhan 430030 Hubei Peoples R;
Huazhong Univ Sci &
Technol Tongji Hosp Hepat Surg Ctr Tongji Med Coll Wuhan Hubei Peoples R;
Vanderbilt Univ Dept Med Nashville TN USA;
Wuhan Univ Renmin Hosp Dept Pancreat Surg 99 Zhangzhidong Rd Wuhan 430030 Hubei Peoples R;
Colorectal cancer; COX and LOX metabolic pathways; Enzyme inhibitors; Apoptosis; Proliferation; PTEN/PI3K/AKT pathway;
机译:COX-2和5-LOX对COX-2和5-LOX的抑制通过促进PTEN和抑制PI3K / AKT途径来调节结肠直肠癌的进展
机译:MiR-494-3P通过调节PTEN / PI3K / AKT途径来促进子宫内膜癌的进展
机译:MicroRNA-155-5P通过PI3K / AKT途径抑制PTEN来促进肝细胞癌进展
机译:Ang-2通过PI3K / Akt途径促进SW1116结肠癌细胞的增殖
机译:DLX4同源蛋白通过上调VEGFA促进ER阴性乳腺癌细胞中的PI3K / Akt抗凋亡途径。
机译:MicroRNA‐155‐5p通过PI3K / Akt途径抑制PTEN促进肝癌进展
机译:IMPH2通过激活PI3K / AKT / MTOR和PI3K / AKT / FOXO1信号通路来促进结肠直肠癌进展