...
机译:ETS1通过UP调节MDR1和MMP9表达促进紫杉醇抗性,激素难治性PC3前列腺癌细胞的化学抑制和侵袭
Department of Urology Gifu University Graduate School of Medicine 1-1 Yanagido Gifu 501-1193;
Department of Longevity and Aging Research Gifu International Institute of Biotechnology 1-1 Naka;
Department of Urology Gifu University Graduate School of Medicine 1-1 Yanagido Gifu 501-1193;
Research Equipment Center Hamamatsu University School of Medicine Hamamatsu Shizuoka 431-3192;
Department of Longevity and Aging Research Gifu International Institute of Biotechnology 1-1 Naka;
Department of Urology Gifu University Graduate School of Medicine 1-1 Yanagido Gifu 501-1193;
Department of Longevity and Aging Research Gifu International Institute of Biotechnology 1-1 Naka;
ETS1; MDR1; Metastasis; MMP9; Paclitaxel resistance; Prostate cancer;
机译:ETS1通过UP调节MDR1和MMP9表达促进紫杉醇抗性,激素难治性PC3前列腺癌细胞的化学抑制和侵袭
机译:miR-148a通过调节MSK1表达,衰减激素耐药性,耐药前列腺癌PC3细胞的紫杉醇抗性
机译:骨桥蛋白和MMP9:与PC3前列腺癌细胞中VEGF表达/分泌和血管生成的关联
机译:截断20-MEL野生型BIM BH3 BOMAIN肽:识别促进前列腺癌(PC3)细胞的细胞死亡所需的最小序列
机译:新型小分子对PC3前列腺癌细胞的细胞毒性作用。
机译:MiR-148a通过调节MSK1表达来减轻激素难治性耐药性前列腺癌PC3细胞对紫杉醇的耐药性
机译:MiR-148a通过调节MSK1表达来减轻激素难治性,耐药性前列腺癌PC3细胞的紫杉醇耐药性*