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Uniform persistence in a prey-predator model with a diseased predator

机译:患有患病捕食者的猎物捕食者模型中的统一持久性

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Following the well-extablished mathematical approach to persistence and its developments contained in Rebelo et al. (Discrete Contin Dyn Syst Ser B 19(4):1155-1170. 10.3934/dcdsb.2014.19.1155, 2014) we give a rigorous theoretical explanation to the numerical results obtained in Bate and Hilker (J Theoret Biol 316:1-8. 10.3934/dcdsb.2014.19.1155, 2013) on a prey-predator Rosenzweig-MacArthur model with functional response of Holling type II, resulting in a cyclic system that is locally unstable, equipped with an infectious disease in the predator population. The proof relies on some repelling conditions that can be applied in an iterative way on a suitable decomposition of the boundary. A full stability analysis is developed, showing how the "invasion condition" for the disease is derived. Some in-depth conclusions and possible further investigations are discussed.
机译:继rebelo等人中持久性的持久性的数学方法和其发展之后。 (离散持续DYN SYST SER B 19(4):1155-1170。10.3934 / DCDSB.2014.19.1155,2014)我们对Bate和Hilker中获得的数值结果提供了严格的理论解释(J Theoret Biol 316:1-8 。10.3934 / DCDSB.2014.19.1155,2011214.19.1155,2011)在捕食性捕食者rosenzweig-macarthur模型,具有荷敏型II型功能响应,导致循环系统,局部不稳定,配备捕食者群体中的传染病。 证明依赖于可以以迭代方式应用的一些排斥条件,以适当的边界分解。 开发了完全稳定性分析,展示了疾病的“侵袭条件”是如何得出的。 讨论了一些深入的结论和可能的进一步调查。

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