Origin of Regular Networks of Joints: Experimental Constraints, Theoretical Background, and Numerical Modeling

摘要

Experimental data show that inelastic straining occurs even at very low pressure before and during brittle fracturing. This process is therefore investigated within the framework of elastoplasticity using 2‐D, three‐layer finite difference modeling. The constitutive model includes both tensile and shear failure mechanisms coupled at the level of the strain softening law. The modeling results show that sets of parallel joints initiate as pure dilation bands, the narrow σ3‐normal bands of localized dilatant damage (inelastic deformation). The band thickness, length, and the initial strain softening degree within it are proportional to the ductility of the material, which increases with the effective stress level (σ1) or pressure. The strength reduction within the bands is accelerated at a certain stage, and the strength locally reaches zero resulting in fracture initiation. The initial fracture then propagates in Mode I following the propagating band. The fracture (joint) appears thus as a band of damaged material with the increased porosity, which is maximum along the axial zone of the band where the material is completely broken. The damage is due to both tensile and shear mechanisms. The role of shear failure increases with the ductility (pressure) increase, which also leads to the band thickness increase. These processes can result in small (band thickness)‐scale oblique shear fractures within the band, causing the increase in the roughness of fracture walls organized in plumose patterns typical of both natural and experimentally generated joints.