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机译:Sunitinib在鼻咽癌和肝癌细胞中诱导NK-Kapp B-依赖性NKG2D配体表达
Southern Med Univ Zhujiang Hosp Dept Hematol Guangzhou 510282 Guangdong Peoples R China;
New York State Univ Inst Genom &
Multiscale Biol Mt Sinai Sch Med New York NY USA;
Southern Med Univ Zhujiang Hosp Dept Hematol Guangzhou 510282 Guangdong Peoples R China;
Southern Med Univ Zhujiang Hosp Dept Hematol Guangzhou 510282 Guangdong Peoples R China;
Southern Med Univ Zhujiang Hosp Dept Hematol Guangzhou 510282 Guangdong Peoples R China;
Southern Med Univ Zhujiang Hosp Dept Hematol Guangzhou 510282 Guangdong Peoples R China;
Southern Med Univ Zhujiang Hosp Dept Hematol Guangzhou 510282 Guangdong Peoples R China;
multitargeted tyrosine kinase inhibitor; sunitinib; NK cells; NKG2DL; NF-kappa b;
机译:Sunitinib在鼻咽癌和肝癌细胞中诱导NK-Kapp B-依赖性NKG2D配体表达
机译:索拉非尼和舒尼替尼在高表达ABCG2的鼻咽癌中诱导NKG2D配体表达的作用 sub>
机译:p38 MAPK抑制剂SB203580通过配体依赖性芳烃受体激活,在鼠和人肝癌细胞系中诱导细胞色素P450 1A1基因表达。
机译:鼻咽癌中的ELF-1表达促进通过调节CCL2 / CCR2信号传导的癌细胞增殖和转移
机译:致瘤性腺病毒12细胞通过减少NKG2D配体的表达逃避NK细胞裂解
机译:细胞因子诱导的杀伤细胞通过NKG2D配体识别有效杀死鼻咽癌的干样癌细胞
机译:细胞因子诱导的杀手细胞通过NKG2D-配体识别有效地杀死鼻咽癌的干燥癌细胞