PDK1 promotes apoptosis of chondrocytes via modulating MAPK pathway in osteoarthritis

Qingtian Ge; Hailin Wang; Xinbao Xu; Libin Xu; Leilei Zhai; Ran Tao

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PDK1 promotes apoptosis of chondrocytes via modulating MAPK pathway in osteoarthritis

机译：PDK1通过调节骨关节炎的Mapk途径促进软骨细胞的凋亡

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来源
《Tissue and Cell》 |2017年第6期|共7页
作者
Qingtian Ge; Hailin Wang; Xinbao Xu; Libin Xu; Leilei Zhai; Ran Tao;
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作者单位

Affiliated Hospital of Nantong University Department of Orthopaedics Nantong University;

Affiliated Hospital of Nantong University Department of Orthopaedics Nantong University;

Affiliated Hospital of Nantong University Department of Orthopaedics Nantong University;

Affiliated Hospital of Nantong University Department of Orthopaedics Nantong University;

Department of Orthopaedics The people Hospital of Jingjiang;

Affiliated Hospital of Nantong University Department of Orthopaedics Nantong University;

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原文格式 PDF
正文语种 other
中图分类细胞生物学;
关键词
PDK1; Osteoarthritis apoptosis; Mapk; Interleukin-1amp; amp; 尾;

机译：PDK1;骨关节炎细胞凋亡;MAPK;白细胞介素-1＆amp;amp;尾;

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专利

1. PDK1 promotes apoptosis of chondrocytes via modulating MAPK pathway in osteoarthritis [J] . Qingtian Ge, Hailin Wang, Xinbao Xu, Tissue and Cell . 2017,第6期

机译：PDK1通过调节骨关节炎的Mapk途径促进软骨细胞的凋亡
2. Effect of luteolin on apoptosis, MAPK and JNK signaling pathways in guinea pig chondrocyte with osteoarthritis [J] . Xue Jianli, Ye Jintao, Xia Zhuqing, Cellular and molecular biology . 2019,第6期

机译：叶英莲对豚鼠软骨细胞凋亡，MAPK和JNK信号通路与骨关节炎的影响
3. The Transient Receptor Potential Channel, Vanilloid 5, Induces Chondrocyte Apoptosis via Ca2+ CaMKII–Dependent MAPK and Akt/ mTOR Pathways in a Rat Osteoarthritis Model [J] . Wei Y., Jin Z., Zhang H., Cellular Physiology and Biochemistry . 2018,第5期

机译：瞬时受体电位通道，香草酸5，在大鼠骨关节炎模型中通过Ca2 + CaMKII依赖性MAPK和Akt / mTOR途径诱导软骨细胞凋亡
4. Modeling PI3K/PDK1/Akt and MAPK Signaling Pathways Using Continuous Petri Nets [C] . Giulia Russo, Marzio Pennisi, Roberta Boscarino, International conference on intelligent computing . 2017

机译：使用连续Petri网建模PI3K / PDK1 / Akt和MAPK信号通路
5. The receptor for advanced glycation endproducts and S100A11 modulate pathologic chondrocyte differentiation and dysregulated cartilage matrix catabolism in osteoarthritis [D] . Cecil, Denise L. 2008

机译：晚期糖基化终产物和S100A11的受体调节骨关节炎的病理性软骨细胞分化和软骨基质分解代谢失调
6. PKR Promotes Oxidative Stress and Apoptosis of Human Articular Chondrocytes by Causing Mitochondrial Dysfunction through p38 MAPK Activation—PKR Activation Causes Apoptosis in Human Chondrocytes [O] . Ching-Hou Ma, Chin-Hsien Wu, I-Ming Jou, 2019

机译：PKR通过p38 MAPK激活引起线粒体功能障碍从而促进人关节软骨细胞的氧化应激和凋亡-PKR激活导致人软骨细胞凋亡
7. The Transient Receptor Potential Channel, Vanilloid 5, Induces Chondrocyte Apoptosis via Ca2+ CaMKII–Dependent MAPK and Akt/ mTOR Pathways in a Rat Osteoarthritis Model [O] . Yingliang Wei, Zhaofeng Jin, He Zhang, 2018

机译：瞬态受体电位通道，香草素5，诱导大鼠骨关节炎模型中CA2 + Camkii依赖性MAPK和AKT / MTOR途径的软骨细胞凋亡

PDK1 promotes apoptosis of chondrocytes via modulating MAPK pathway in osteoarthritis

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