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机译:腺病毒12型E1A抑制雄激素受体转基因功能的抑制造型前列腺癌细胞存活
Department of UrologyQilu Hospital of Shandong UniversityJinan P. R. China;
Department of Anatomy and Cell BiologyUniversity of Florida College of MedicineGainesville Florida;
Department of Anatomy and Cell BiologyUniversity of Florida College of MedicineGainesville Florida;
Department of Anatomy and Cell BiologyUniversity of Florida College of MedicineGainesville Florida;
Department of Anatomy and Cell BiologyUniversity of Florida College of MedicineGainesville Florida;
Department of Anatomy and Cell BiologyUniversity of Florida College of MedicineGainesville Florida;
Department of Microbiology and ImmunologyThe University of Western OntarioOntario Canada;
Laboratory of Molecular EndocrinologyKU LeuvenLeuven Belgium;
Masonic Cancer CenterUniversity of MinnesotaMinneapolis Minnesota;
Department of Anatomy and Cell BiologyUniversity of Florida College of MedicineGainesville Florida;
Department of Anatomy and Cell BiologyUniversity of Florida College of MedicineGainesville Florida;
androgen receptor; AR splice variants (AR‐Vs); PI3K‐kinase (PI3K)‐Akt‐mTOR signaling; prostate cancer gene therapy; transcriptional regulation;
机译:腺病毒12型E1A抑制雄激素受体转基因功能的抑制造型前列腺癌细胞存活
机译:Src激酶增强雄激素受体的反式激活功能和非雄激素依赖性前列腺癌C4-2细胞的侵袭
机译:FOXO1的转录非依赖性功能可抑制前列腺癌细胞中雄激素非依赖性的雄激素受体活化。
机译:雄激素受体和PI3K / AKT在雄激素难治性前列腺癌细胞存活中的作用
机译:Stat5a / b在晚期前列腺癌中的治疗靶点:Stat5a / b的抑制作用通过依赖于雄激素受体的机制来抑制前列腺癌的生长。
机译:腺病毒12型E1A抑制雄激素受体反式激活功能破坏了前列腺癌细胞的存活
机译:Transgelin通过抑制ARA54增强的雄激素受体反式激活和前列腺癌细胞的生长而起抑制作用。
机译:新型抗β2-微球蛋白抗体抑制前列腺癌细胞中雄激素受体的表达,存活和进展。