...
机译:PI3K / AKT途径参与抑制SASH1通过下调SHH-GLIL信号抑制SASH1诱导的肝癌细胞侵袭和转移
Zhengzhou Univ Affiliated Hosp 1 1 East Jianshe Rd Zhengzhou 450052 Henan Peoples R China;
Zhengzhou Univ Affiliated Hosp 1 1 East Jianshe Rd Zhengzhou 450052 Henan Peoples R China;
Zhengzhou Univ Affiliated Hosp 1 1 East Jianshe Rd Zhengzhou 450052 Henan Peoples R China;
Zhengzhou Univ Affiliated Hosp 1 1 East Jianshe Rd Zhengzhou 450052 Henan Peoples R China;
Zhengzhou Univ Affiliated Hosp 1 1 East Jianshe Rd Zhengzhou 450052 Henan Peoples R China;
SASH1; Hepatocarcinoma; Invasion; Metastasis; Shh-Glil; PI3K-AKT;
机译:PI3K / AKT途径参与抑制SASH1通过下调SHH-GLIL信号抑制SASH1诱导的肝癌细胞侵袭和转移
机译:抑制脂肪酸合酶通过下调HER2 / PI3K / AKT信号转导通路的活性来抑制U-2 OS细胞的侵袭和迁移
机译:LNCRNA MEG3通过调节PI3K-AKT-MTOR和BECLIN-1信号通路通过下调的ILF 3和自噬抑制有助于肝癌HEPG2细胞中的腺苷诱导的细胞毒性。
机译:PI3K / AKT /GSK3β途径参与神经元和胶质细胞的光动力损伤
机译:多靶点受体酪氨酸激酶抑制剂利尼法尼(ABT-869)通过磷酸肌醇3激酶(PI3K)/ AKT依赖性信号传导途径诱导凋亡并抑制白血病细胞的增殖。
机译:SASH1通过PI3K / Akt信号通路抑制甲状腺癌细胞的增殖和侵袭
机译:Aclidinium通过下调PI3K / AKT / MTOR信号通路抑制卵巢癌SKOV3细胞的增殖和转移