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机译:PAR2抑制增强了结直肠癌细胞对5-FU的敏感性,减少了EMT信号传导
Soochow Univ Dept Pathol Affiliated Hosp 1 899 Pinghai Rd Suzhou 215006 Jiangsu Peoples R;
Soochow Univ Dept Gen Surg Affiliated Hosp 2 Suzhou Jiangsu Peoples R China;
Nanjing Med Univ Dept Oncol Affiliated Canc Hosp Nanjing Jiangsu Peoples R China;
Soochow Univ Dept Oncol Affiliated Hosp 1 Suzhou Jiangsu Peoples R China;
Soochow Univ Dept Pathol Affiliated Hosp 1 899 Pinghai Rd Suzhou 215006 Jiangsu Peoples R;
Colorectal cancer (CRC); Protease-activated receptor 2 (PAR2); 5-Fluorouracil (5-FU); Transforming growth factor-beta (TGF-beta);
机译:PAR2抑制增强了结直肠癌细胞对5-FU的敏感性,减少了EMT信号传导
机译:( - ) - EpigallocateChin Gallate(EGCG)通过抑制GRP78 / NF-Kappa B / MDR1途径增强结肠直肠癌细胞至5-FU的敏感性
机译:ID4通过CK18相关的AKT和EMT信号传导抑制结直肠癌HCT116细胞的生长和侵袭
机译:5-FU的组合施用CDHP施加5-FU耐胃癌细胞的协同抗肿瘤效果
机译:差分P53信号响应于5-fu和依托磷脂在癌症治疗中通过DPYD和CHK2调节毒性
机译:ID4通过CK18相关的AKT和EMT信号传导抑制结直肠癌HCT116细胞的生长和侵袭
机译:黄芪甲苷诱导miR-134表达通过抑制结肠直肠癌细胞系sW-480中的CREB1信号,降低EmT并提高化疗敏感性