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机译:回复康和布鲁克斯:评论PRA1的结合的解释,真菌免疫逃离蛋白从念珠菌蛋白到人C3以及C3激活时的构象变化
Department of Infection Biology Leibniz-Institute for Natural Product Research and Infection;
Department of Infection Biology Leibniz-Institute for Natural Product Research and Infection;
Department of Infection Biology Leibniz-Institute for Natural Product Research and Infection;
Department of Infection Biology Leibniz-Institute for Natural Product Research and Infection;
University of Würzburg Institute for Virology and Immunobiology;
Friedrich-Schiller-University;
Institute of Medical Microbiology Medical School Hannover;
Department of Infection Biology Leibniz-Institute for Natural Product Research and Infection;
机译:回复康和布鲁克斯:评论PRA1的结合的解释,真菌免疫逃离蛋白从念珠菌蛋白到人C3以及C3激活时的构象变化
机译:白色念珠菌的pH调节抗原1(Pra1)优先靶向并灭活人类补体C3和C3激活片段以逃避宿主免疫攻击
机译:白色念珠菌的pH调节抗原1(Pra1)裂解人补体成分C3,并阻断C3a介导的抗真菌活性和效应子功能。
机译:免疫融合蛋白对白色念珠菌的结合亲和力和抗真菌活性
机译:两种免疫逃避蛋白Gpm1和Pra1的序列变异和蛋白表达水平影响临床白色念珠菌分离株的毒力。
机译:两种免疫逃逸蛋白Gpm1和pra1的序列变异和蛋白质表达水平影响临床白色念珠菌分离株的毒力。