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机译:在肝纤维化进展过程中,肝脏蛋白表达降低了肝脏
Capital Med Univ Beijing Friendship Hosp Dept Infect Beijing 100050 Peoples R China;
Capital Med Univ Beijing Friendship Hosp Dept Infect Beijing 100050 Peoples R China;
Capital Med Univ Beijing Friendship Hosp Dept Infect Beijing 100050 Peoples R China;
Capital Med Univ Beijing Friendship Hosp Dept Infect Beijing 100050 Peoples R China;
Capital Med Univ Beijing Friendship Hosp Dept Infect Beijing 100050 Peoples R China;
Capital Med Univ Beijing Friendship Hosp Dept Infect Beijing 100050 Peoples R China;
Capital Med Univ Beijing Youan Hosp Gastroenterol &
Hepatol Dept 8 Youan Men Wai Xi Tou Tiao Rd;
Capital Med Univ Beijing Obstet &
Gynecol Hosp Dept Internal Med 251 Yaojiayuan Rd Beijing;
Arkadia; hepatic fibrosis; E3 ubiquitin ligase; transforming growth factor-beta 1 signaling pathway; post-translational modification;
机译:肝纤维化进展过程中肝脏肝脏Arkadia蛋白表达降低
机译:在肝纤维化进展过程中,肝脏蛋白表达降低了肝脏
机译:Crassostrea gigas的蛋白质提取物通过减少肝脏组织中CTGF,TGF-beta 1和NF-κB的表达来减轻CCl4诱导的大鼠肝纤维化。
机译:利用肝纤维化进展和组织学模型评估由纤维结构和组织粘度引起的剪切波弥散
机译:血小板源性生长因子受体α在慢性肝损伤中的新兴作用:肝纤维化的潜在治疗靶点。
机译:肝纤维化进展过程中肝脏肝脏Arkadia蛋白表达降低
机译:大鼠肝脏纤维化自发消退的机制。肝星状细胞凋亡和金属蛋白酶抑制剂的肝表达降低。