...
机译:通过PI3K / AKT / MTOR信号通路抑制细胞凋亡和自噬激活,Notoginesene R1衰减葡萄糖诱导的泛键细胞损伤
Guangxi Univ Chinese Med Ruikang Hosp Dept Nephrol Nanning 530011 Guangxi Peoples R China;
Sichuan Acad Med Sci Dept Gynecol Chengdu 610072 Sichuan Peoples R China;
Guangxi Univ Chinese Med Sch Basic Med Sci Nanning 530001 Guangxi Peoples R China;
Guangxi Univ Chinese Med Sch Basic Med Sci Nanning 530001 Guangxi Peoples R China;
Univ Elect Sci &
Technol China Dept Biomed Engn Sch Med Chengdu 610054 Sichuan Peoples R China;
Guangxi Univ Chinese Med Ruikang Hosp Dept Nephrol Nanning 530011 Guangxi Peoples R China;
Guangxi Univ Chinese Med Ruikang Hosp Dept Nephrol Nanning 530011 Guangxi Peoples R China;
Guangxi Univ Chinese Med Ruikang Hosp Dept Nephrol Nanning 530011 Guangxi Peoples R China;
Nationwide Childrens Hosp Res Inst Ctr Perinatal Res 575 Childrens Crossrd Columbus OH 43215;
Sichuan Prov Peoples Hosp 32 First Ring Rd Chengdu 610072 Sichuan Peoples R China;
podocyte; apoptosis; autophagy; notoginsenoside R1; PI3K/Akt/mTOR pathway;
机译:三七皂苷R1通过抑制凋亡和通过PI3K / Akt / mTOR信号通路的自噬激活来减轻葡萄糖诱导的足细胞损伤
机译:三七皂甙R1通过激活PI3K / Akt信号通路改善糖尿病肾病大鼠的足细胞损伤
机译:三七皂甙R1通过激活PI3K / Akt信号通路改善糖尿病肾病大鼠的足细胞损伤
机译:阳离子聚苯乙烯纳米球通过抑制Akt / mTOR和激活巨噬细胞和上皮细胞中的AMPK信号通路来诱导自噬
机译:PI3k / AKT / mTOR信号通路的遗传分析。
机译:三七皂苷R1通过抑制细胞凋亡和通过PI3K / Akt / mTOR信号通路激活自噬减轻葡萄糖诱导的足细胞损伤
机译:Notoginsenide R1通过抑制细胞凋亡和通过PI3K / AKT / MTOR信号传导途径抑制血糖诱导的葡萄糖诱导的泛骨细胞损伤