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机译:miR-134 miR-134靶向pdcd7 pdcd7,以减少E-cadherin表达并增强口腔癌进展
Institute of Oral BiologySchool of Dentistry National Yang‐Ming UniversityTaipei Taiwan;
Department of DentistrySchool of Dentistry National Yang‐Ming UniversityTaipei Taiwan;
Institute of Oral BiologySchool of Dentistry National Yang‐Ming UniversityTaipei Taiwan;
Department of DentistrySchool of Dentistry National Yang‐Ming UniversityTaipei Taiwan;
Department of DentistrySchool of Dentistry National Yang‐Ming UniversityTaipei Taiwan;
Institute of Oral BiologySchool of Dentistry National Yang‐Ming UniversityTaipei Taiwan;
Institute of Oral BiologySchool of Dentistry National Yang‐Ming UniversityTaipei Taiwan;
carcinoma; E‐cadherin; miRNA; miR‐134; PDCD7;
机译:miR-134 miR-134靶向pdcd7 pdcd7,以减少E-cadherin表达并增强口腔癌进展
机译:miR-134的过度表达通过下调abcc1表达增强了乳腺癌细胞对多柔比星的敏感性
机译:黄芪甲苷IV诱导的miR-134表达通过抑制结直肠癌细胞系SW-480中的CREB1信号传导而降低EMT并增加化学敏感性。
机译:miR-9,miR-21,miR-155和miR-944的上调及其与福尔马林固定石蜡嵌入宫颈癌样品的HPV E6 / E7表达的相关性
机译:miR-223双链体的双链(miR-223-5p和miR-223-3p)抑制癌细胞侵袭性:靶向基因参与膀胱癌的发病机理
机译:通过针对启动子的miR-373增强E-cadherin表达抑制膀胱癌细胞的生长和转移
机译:黄芪甲苷诱导miR-134表达通过抑制结肠直肠癌细胞系sW-480中的CREB1信号,降低EmT并提高化疗敏感性